Pages that link to "Q28586378"
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The following pages link to Decreases in the precision of Purkinje cell pacemaking cause cerebellar dysfunction and ataxia (Q28586378):
Displaying 50 items.
- Calcium channel, voltage-dependent, alpha 2/delta subunit 2 (Q21981686) (← links)
- Calcium channel, voltage-dependent, P/Q type, alpha 1A subunit (Q21984376) (← links)
- Selective positive modulation of the SK3 and SK2 subtypes of small conductance Ca2 -activated K channels (Q24564966) (← links)
- Precise control of movement kinematics by optogenetic inhibition of Purkinje cell activity (Q24608924) (← links)
- The cerebellum and migraine (Q24628942) (← links)
- Strength and timing of motor responses mediated by rebound firing in the cerebellar nuclei after Purkinje cell activation (Q24630442) (← links)
- The ducky(2J) mutation in Cacna2d2 results in reduced spontaneous Purkinje cell activity and altered gene expression (Q24676262) (← links)
- Therapeutic Approaches to Genetic Ion Channelopathies and Perspectives in Drug Discovery (Q26744730) (← links)
- Precision medicine in spinocerebellar ataxias: treatment based on common mechanisms of disease (Q26764958) (← links)
- Integration of modeling with experimental and clinical findings synthesizes and refines the central role of inositol 1,4,5-trisphosphate receptor 1 in spinocerebellar ataxia (Q26864714) (← links)
- Toll-Like Receptor 4 Deficiency Impairs Motor Coordination (Q27313529) (← links)
- The riluzole derivative 2-amino-6-trifluoromethylthio-benzothiazole (SKA-19), a mixed KCa2 activator and NaV blocker, is a potent novel anticonvulsant (Q27339255) (← links)
- Disruption of learned timing in P/Q calcium channel mutants (Q27438153) (← links)
- The physiological basis of therapies for cerebellar ataxias (Q28075917) (← links)
- Climbing fiber discharge regulates cerebellar functions by controlling the intrinsic characteristics of purkinje cell output (Q28286052) (← links)
- Major channels involved in neuropsychiatric disorders and therapeutic perspectives (Q28290639) (← links)
- Which medication do I need to manage dizzy patients? (Q28300141) (← links)
- Neuroprotective role of liver growth factor "LGF" in an experimental model of cerebellar ataxia (Q28397547) (← links)
- Targeted deletion of a single Sca8 ataxia locus allele in mice causes abnormal gait, progressive loss of motor coordination, and Purkinje cell dendritic deficits (Q28504521) (← links)
- SK2 channel expression and function in cerebellar Purkinje cells (Q28573148) (← links)
- A defined heteromeric KV1 channel stabilizes the intrinsic pacemaking and regulates the output of deep cerebellar nuclear neurons to thalamic targets (Q28581867) (← links)
- Pharmacological gating modulation of small- and intermediate-conductance Ca(2 )-activated K( ) channels (KCa2.x and KCa3.1) (Q28833834) (← links)
- Dominant-negative suppression of Cav2.1 currents by alpha(1)2.1 truncations requires the conserved interaction domain for beta subunits (Q30429315) (← links)
- Studying Cerebellar Circuits by Remote Control of Selected Neuronal Types with GABA(A) Receptors (Q30483976) (← links)
- Synaptic inhibition of Purkinje cells mediates consolidation of vestibulo-cerebellar motor learning. (Q30489167) (← links)
- Reduced nucleus accumbens SK channel activity enhances alcohol seeking during abstinence. (Q30493928) (← links)
- KCa channels as therapeutic targets in episodic ataxia type-2 (Q30495677) (← links)
- The therapeutic mode of action of 4-aminopyridine in cerebellar ataxia (Q30495678) (← links)
- Delayed postnatal loss of P/Q-type calcium channels recapitulates the absence epilepsy, dyskinesia, and ataxia phenotypes of genomic Cacna1a mutations. (Q30499188) (← links)
- SK2 Channels Are Neuroprotective for Ischemia-Induced Neuronal Cell Death (Q30512907) (← links)
- Alterations in cerebellar physiology are associated with a stiff-legged gait in Atcay(ji-hes) mice (Q30580787) (← links)
- BK channels control cerebellar Purkinje and Golgi cell rhythmicity in vivo (Q33517128) (← links)
- CaV2.1 channelopathies (Q33537218) (← links)
- Molecular and cellular basis of small--and intermediate-conductance, calcium-activated potassium channel function in the brain (Q33560844) (← links)
- Compensatory regulation of Cav2.1 Ca2 channels in cerebellar Purkinje neurons lacking parvalbumin and calbindin D-28k (Q33593543) (← links)
- Quantitative localization of Cav2.1 (P/Q-type) voltage-dependent calcium channels in Purkinje cells: somatodendritic gradient and distinct somatic coclustering with calcium-activated potassium channels. (Q33652631) (← links)
- Calcium channels and synaptic transmission in familial hemiplegic migraine type 1 animal models. (Q33680269) (← links)
- A positive feedback loop linking enhanced mGluR function and basal calcium in spinocerebellar ataxia type 2 (Q33728850) (← links)
- Loss of beta-III spectrin leads to Purkinje cell dysfunction recapitulating the behavior and neuropathology of spinocerebellar ataxia type 5 in humans. (Q33801655) (← links)
- Disruption of the olivo-cerebellar circuit by Purkinje neuron-specific ablation of BK channels (Q33982019) (← links)
- The action potential in mammalian central neurons (Q34004305) (← links)
- Systematic regional variations in Purkinje cell spiking patterns (Q34075631) (← links)
- Physiologic changes associated with cerebellar dystonia (Q34145378) (← links)
- In vivo evidence that TRAF4 is required for central nervous system myelin homeostasis (Q34170353) (← links)
- A new Kv1.2 channelopathy underlying cerebellar ataxia (Q34186439) (← links)
- Intrinsic plasticity complements long-term potentiation in parallel fiber input gain control in cerebellar Purkinje cells. (Q34262354) (← links)
- Computational analysis of calcium signaling and membrane electrophysiology in cerebellar Purkinje neurons associated with ataxia (Q34306449) (← links)
- The first knockin mouse model of episodic ataxia type 2 (Q34325294) (← links)
- The juvenile myoclonic epilepsy mutant of the calcium channel β(4) subunit displays normal nuclear targeting in nerve and muscle cells (Q34373916) (← links)
- p75 regulates Purkinje cell firing by modulating SK channel activity through Rac1. (Q34467645) (← links)