Pages that link to "Q28510123"
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The following pages link to CaMKII binding to GluN2B is critical during memory consolidation (Q28510123):
Displaying 50 items.
- Calcium/calmodulin-dependent protein kinase II alpha (Q21496046) (← links)
- Glutamate receptor, ionotropic, NMDA2B (epsilon 2) (Q21496048) (← links)
- Discs large MAGUK scaffold protein 4 (Q21498713) (← links)
- Distinct roles of multiple isoforms of CaMKII in signaling to the nucleus (Q26823646) (← links)
- Protein degradation and protein synthesis in long-term memory formation (Q26830414) (← links)
- Calcium dysregulation in atrial fibrillation: the role of CaMKII (Q27027326) (← links)
- Surface expression of hippocampal NMDA GluN2B receptors regulated by fear conditioning determines its contribution to memory consolidation in adult rats (Q27321058) (← links)
- Molecular and genetic determinants of the NMDA receptor for superior learning and memory functions (Q27332137) (← links)
- Capping of the N-terminus of PSD-95 by calmodulin triggers its postsynaptic release (Q27683002) (← links)
- Molecular mechanisms of synaptic remodeling in alcoholism (Q28080843) (← links)
- Protection of α-CaMKII from Dephosphorylation by GluN2B Subunit of NMDA Receptor Is Abolished by Mutation of Glu96 or His282 of α-CaMKII (Q28554007) (← links)
- Surface dynamics of GluN2B-NMDA receptors controls plasticity of maturing glutamate synapses (Q28581987) (← links)
- The CaMKII holoenzyme structure in activation-competent conformations (Q30855420) (← links)
- Multiple spatial and kinetic subpopulations of CaMKII in spines and dendrites as resolved by single-molecule tracking PALM. (Q33671699) (← links)
- Autonomous CaMKII requires further stimulation by Ca2 /calmodulin for enhancing synaptic strength (Q33913227) (← links)
- Proteomic identification of altered cerebral proteins in the complex regional pain syndrome animal model. (Q34275621) (← links)
- Effect of multimeric structure of CaMKII in the GluN2B-mediated modulation of kinetic parameters of ATP. (Q34427989) (← links)
- On the mechanism of synaptic depression induced by CaMKIIN, an endogenous inhibitor of CaMKII. (Q34474360) (← links)
- NMDA receptor subunit diversity: impact on receptor properties, synaptic plasticity and disease (Q34651972) (← links)
- SynGAP regulates protein synthesis and homeostatic synaptic plasticity in developing cortical networks (Q35082678) (← links)
- CaMKII binding to GluN2B is differentially affected by macromolecular crowding reagents (Q35163005) (← links)
- Subcellular organization of camkii in rat hippocampal pyramidal neurons. (Q35538777) (← links)
- Substrate-selective and calcium-independent activation of CaMKII by α-actinin (Q38630004) (← links)
- Competition between α-actinin and Ca²⁺-calmodulin controls surface retention of the L-type Ca²⁺ channel Ca(V)1.2. (Q36062292) (← links)
- Functional and mechanistic exploration of an adult neurogenesis-promoting small molecule. (Q36117399) (← links)
- Bi-directional regulation of CaMKIIα phosphorylation at Thr286 by NMDA receptors in cultured cortical neurons (Q36210224) (← links)
- CaMKII regulation in information processing and storage (Q36287751) (← links)
- Notch1 Regulates Hippocampal Plasticity Through Interaction with the Reelin Pathway, Glutamatergic Transmission and CREB Signaling (Q36320082) (← links)
- Coordination of Protein Phosphorylation and Dephosphorylation in Synaptic Plasticity (Q36323396) (← links)
- CaMKII regulates proteasome phosphorylation and activity and promotes memory destabilization following retrieval. (Q36581669) (← links)
- The CaMKII/NMDAR complex as a molecular memory (Q36639359) (← links)
- Atrial remodelling in atrial fibrillation: CaMKII as a nodal proarrhythmic signal (Q36650585) (← links)
- Breaking it down: the ubiquitin proteasome system in neuronal morphogenesis (Q36651360) (← links)
- Activated CaMKII couples GluN2B and casein kinase 2 to control synaptic NMDA receptors (Q36736218) (← links)
- Delay-dependent impairment of spatial working memory with inhibition of NR2B-containing NMDA receptors in hippocampal CA1 region of rats (Q36741422) (← links)
- Behavioral and structural responses to chronic cocaine require a feedforward loop involving ΔFosB and calcium/calmodulin-dependent protein kinase II in the nucleus accumbens shell (Q36856397) (← links)
- Persistent reversal of enhanced amphetamine intake by transient CaMKII inhibition (Q37008465) (← links)
- Long-term potentiation: peeling the onion (Q37033863) (← links)
- Enzymatic activity of CaMKII is not required for its interaction with the glutamate receptor subunit GluN2B. (Q37325643) (← links)
- Involvement of Brain-Enriched Guanylate Kinase-Associated Protein (BEGAIN) in Chronic Pain after Peripheral Nerve Injury. (Q37343075) (← links)
- GluN2A and GluN2B subunit-containing NMDA receptors in hippocampal plasticity. (Q37350719) (← links)
- Phosphorylation of Ser1166 on GluN2B by PKA is critical to synaptic NMDA receptor function and Ca2 signaling in spines (Q37479508) (← links)
- Fluoxetine epigenetically alters the CaMKIIα promoter in nucleus accumbens to regulate ΔFosB binding and antidepressant effects (Q37642433) (← links)
- Phosphorylation of Ser1928 mediates the enhanced activity of the L-type Ca2 channel Cav1.2 by the β2-adrenergic receptor in neurons (Q37643798) (← links)
- Infralimbic BDNF/TrkB enhancement of GluN2B currents facilitates extinction of a cocaine-conditioned place preference (Q37717664) (← links)
- A frontier in the understanding of synaptic plasticity: solving the structure of the postsynaptic density. (Q38005085) (← links)
- AMPARs and synaptic plasticity: the last 25 years (Q38158723) (← links)
- Roles of subunit phosphorylation in regulating glutamate receptor function. (Q38167533) (← links)
- The synaptic plasticity and memory hypothesis: encoding, storage and persistence (Q38168197) (← links)
- CaMKII: claiming center stage in postsynaptic function and organization (Q38181739) (← links)