Pages that link to "Q28507277"
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The following pages link to Essential roles for Fe65, Alzheimer amyloid precursor-binding protein, in the cellular response to DNA damage (Q28507277):
Displaying 32 items.
- Amyloid beta (A4) precursor protein (Q14865884) (← links)
- Amyloid beta (A4) precursor protein-binding, family B, member 1 (Q21498719) (← links)
- Regulation of FE65 nuclear translocation and function by amyloid beta-protein precursor in osmotically stressed cells (Q24311363) (← links)
- Tyrosine dephosphorylation of H2AX modulates apoptosis and survival decisions (Q24313501) (← links)
- Fe65 is required for Tip60-directed histone H4 acetylation at DNA strand breaks (Q28575987) (← links)
- Increased origin activity in transformed versus normal cells: identification of novel protein players involved in DNA replication and cellular transformation (Q33523235) (← links)
- A novel function of the Fe65 neuronal adaptor in estrogen receptor action in breast cancer cells (Q33556060) (← links)
- Tyr(682) in the intracellular domain of APP regulates amyloidogenic APP processing in vivo (Q33754509) (← links)
- Identification and characterization of a neuronal enriched novel transcript encoding the previously described p60Fe65 isoform (Q33985290) (← links)
- A set of miRNAs participates in the cellular senescence program in human diploid fibroblasts. (Q34229984) (← links)
- Fe65 Suppresses Breast Cancer Cell Migration and Invasion through Tip60 Mediated Cortactin Acetylation (Q35844455) (← links)
- The mitochondria-targeted antioxidant MitoQ prevents loss of spatial memory retention and early neuropathology in a transgenic mouse model of Alzheimer's disease. (Q35908404) (← links)
- Fe65 Is Phosphorylated on Ser289 after UV-Induced DNA Damage (Q36016912) (← links)
- Oxidative stress activates a positive feedback between the gamma- and beta-secretase cleavages of the beta-amyloid precursor protein (Q36417576) (← links)
- Brain ischemia activates β- and γ-secretase cleavage of amyloid precursor protein: significance in sporadic Alzheimer's disease (Q36511844) (← links)
- Connecting chromatin modifying factors to DNA damage response (Q36658638) (← links)
- Immune response profiling identifies autoantibodies specific to Moyamoya patients (Q36828028) (← links)
- Evidence that the Amyloid beta Precursor Protein-intracellular domain lowers the stress threshold of neurons and has a "regulated" transcriptional role (Q36882015) (← links)
- Regulation of amyloid beta-protein precursor by phosphorylation and protein interactions (Q37225056) (← links)
- Chromatin modifications associated with DNA double-strand breaks repair as potential targets for neurological diseases (Q37249162) (← links)
- Ubiquitylation of Fe65 adaptor protein by neuronal precursor cell expressed developmentally down regulated 4-2 (Nedd4-2) via the WW domain interaction with Fe65. (Q37337899) (← links)
- Amyloid beta a4 precursor protein-binding family B member 1 (FE65) interactomics revealed synaptic vesicle glycoprotein 2A (SV2A) and sarcoplasmic/endoplasmic reticulum calcium ATPase 2 (SERCA2) as new binding proteins in the human brain (Q37563893) (← links)
- UV irradiation accelerates amyloid precursor protein (APP) processing and disrupts APP axonal transport. (Q37603784) (← links)
- Amyloid-β Production: Major Link Between Oxidative Stress and BACE1 (Q37946306) (← links)
- Fe65 matters: new light on an old molecule (Q38057997) (← links)
- An epigenomic role of Fe65 in the cellular response to DNA damage (Q38459516) (← links)
- Roles of the intramolecular regions of FE65 in its trans-accumulation and in p53 stabilization in the nuclear matrix of osmotically stressed cells (Q39758621) (← links)
- Oxidative stress mediates the pathogenic effect of different Alzheimer's disease risk factors (Q40683231) (← links)
- The amyloid precursor protein intracellular domain-fe65 multiprotein complexes: a challenge to the amyloid hypothesis for Alzheimer's disease? (Q41899169) (← links)
- Complexity and Selectivity of γ-Secretase Cleavage on Multiple Substrates: Consequences in Alzheimer's Disease and Cancer. (Q47441289) (← links)
- A differential proteomic approach reveals an evolutionary conserved regulation of Nme proteins by Fe65 in C. elegans and mouse. (Q51959968) (← links)
- Fe65 Ser228 is phosphorylated by ATM/ATR and inhibits Fe65-APP-mediated gene transcription. (Q54309703) (← links)