Pages that link to "Q28284342"
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The following pages link to TLS facilitates transport of mRNA encoding an actin-stabilizing protein to dendritic spines (Q28284342):
Displaying 50 items.
- Chronic cocaine-regulated epigenomic changes in mouse nucleus accumbens. (Q21999524) (← links)
- Fused in sarcoma (FUS) interacts with the cytolinker protein plectin: implications for FUS subcellular localization and function (Q24301732) (← links)
- A conserved N-terminal motif is required for complex formation between FUS, EWSR1, TAF15 and their oncogenic fusion proteins (Q24319718) (← links)
- Abundant FUS-immunoreactive pathology in neuronal intermediate filament inclusion disease (Q24628685) (← links)
- ALS-associated fused in sarcoma (FUS) mutations disrupt Transportin-mediated nuclear import (Q24630100) (← links)
- A new subtype of frontotemporal lobar degeneration with FUS pathology (Q24647697) (← links)
- TLS inhibits RNA polymerase III transcription (Q24654017) (← links)
- Dysregulated axonal RNA translation in amyotrophic lateral sclerosis (Q26753110) (← links)
- FUS-mediated regulation of alternative RNA processing in neurons: insights from global transcriptome analysis (Q26773038) (← links)
- Alterations in stress granule dynamics driven by TDP-43 and FUS: a link to pathological inclusions in ALS? (Q26777971) (← links)
- Molecular motor proteins and amyotrophic lateral sclerosis (Q26820831) (← links)
- Cytoplasmic sequestration of FUS/TLS associated with ALS alters histone marks through loss of nuclear protein arginine methyltransferase 1. (Q27301265) (← links)
- Early lethality and neuronal proteinopathy in mice expressing cytoplasm-targeted FUS that lacks the RNA recognition motif. (Q27334411) (← links)
- FUS transgenic rats develop the phenotypes of amyotrophic lateral sclerosis and frontotemporal lobar degeneration (Q27342067) (← links)
- Transportin1: a marker of FTLD-FUS (Q28245695) (← links)
- TDP-43 and FUS in amyotrophic lateral sclerosis and frontotemporal dementia (Q28294001) (← links)
- FUS-NLS/Transportin 1 complex structure provides insights into the nuclear targeting mechanism of FUS and the implications in ALS (Q28484269) (← links)
- Selective frontoinsular von Economo neuron and fork cell loss in early behavioral variant frontotemporal dementia (Q28732862) (← links)
- (Q29039632) (redirect page) (← links)
- FUS is sequestered in nuclear aggregates in ALS patient fibroblasts (Q30458567) (← links)
- Biochemical Properties and Biological Functions of FET Proteins (Q30459373) (← links)
- The ALS disease protein TDP-43 is actively transported in motor neuron axons and regulates axon outgrowth. (Q30523244) (← links)
- Overexpression of human wild-type FUS causes progressive motor neuron degeneration in an age- and dose-dependent fashion (Q30532680) (← links)
- FUS/TLS assembles into stress granules and is a prosurvival factor during hyperosmolar stress (Q30577079) (← links)
- Gene expression responses to FUS, EWS, and TAF15 reduction and stress granule sequestration analyses identifies FET-protein non-redundant functions (Q31104690) (← links)
- The multifunctional FUS, EWS and TAF15 proto-oncoproteins show cell type-specific expression patterns and involvement in cell spreading and stress response (Q33351073) (← links)
- The TET family of proteins: functions and roles in disease (Q33999337) (← links)
- Distinct pathological subtypes of FTLD-FUS. (Q34024578) (← links)
- Multistep process of FUS aggregation in the cell cytoplasm involves RNA-dependent and RNA-independent mechanisms. (Q34155410) (← links)
- The tip of the iceberg: RNA-binding proteins with prion-like domains in neurodegenerative disease. (Q34263380) (← links)
- Fus gene mutations in familial and sporadic amyotrophic lateral sclerosis (Q34265540) (← links)
- Functions of FUS/TLS from DNA repair to stress response: implications for ALS. (Q34305789) (← links)
- Mutant FUS proteins that cause amyotrophic lateral sclerosis incorporate into stress granules (Q34308339) (← links)
- Nuclear localization sequence of FUS and induction of stress granules by ALS mutants. (Q34384744) (← links)
- A network of RNA and protein interactions in Fronto Temporal Dementia (Q35194634) (← links)
- Normal and functional TP53 in genetically stable myxoid/round cell liposarcoma (Q35416886) (← links)
- Linking RNA Dysfunction and Neurodegeneration in Amyotrophic Lateral Sclerosis (Q35501802) (← links)
- Understanding the role of TDP-43 and FUS/TLS in ALS and beyond. (Q35587208) (← links)
- Aging--RNA in development and disease (Q35605882) (← links)
- Motor neuron apoptosis and neuromuscular junction perturbation are prominent features in a Drosophila model of Fus-mediated ALS (Q35887683) (← links)
- Translocated in liposarcoma regulates the distribution and function of mammalian enabled, a modulator of actin dynamics (Q35929154) (← links)
- Altered mRNP granule dynamics in FTLD pathogenesis. (Q38634221) (← links)
- Mutations in FUS cause FALS and SALS in French and French Canadian populations (Q36292149) (← links)
- Entorhinal cortical neurons are the primary targets of FUS mislocalization and ubiquitin aggregation in FUS transgenic rats (Q36317708) (← links)
- TDP-43 and FUS en route from the nucleus to the cytoplasm (Q36336278) (← links)
- Arginine methylation next to the PY-NLS modulates Transportin binding and nuclear import of FUS. (Q36408540) (← links)
- Super-Resolution Microscopy Reveals Presynaptic Localization of the ALS/FTD Related Protein FUS in Hippocampal Neurons (Q36453024) (← links)
- Dendritic mRNA: transport, translation and function (Q36937891) (← links)
- Understanding the importance of mRNA transport in memory. (Q37131111) (← links)
- Amyotrophic lateral sclerosis-linked FUS/TLS alters stress granule assembly and dynamics (Q37153932) (← links)