Pages that link to "Q24537292"
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The following pages link to Structural elements in domain IV that influence biophysical and pharmacological properties of human alpha1A-containing high-voltage-activated calcium channels (Q24537292):
Displaying 39 items.
- Systematic identification of splice variants in human P/Q-type channel alpha1(2.1) subunits: implications for current density and Ca2 -dependent inactivation (Q24321661) (← links)
- The familial hemiplegic migraine mutation R192Q reduces G-protein-mediated inhibition of P/Q-type (Ca(V)2.1) calcium channels expressed in human embryonic kidney cells (Q24657270) (← links)
- From foe to friend: using animal toxins to investigate ion channel function (Q26864410) (← links)
- alpha(1E) subunits form the pore of three cerebellar R-type calcium channels with different pharmacological and permeation properties (Q28378561) (← links)
- Novel Cav2.1 splice variants isolated from Purkinje cells do not generate P-type Ca2 current (Q28585263) (← links)
- Transcript scanning reveals novel and extensive splice variations in human l-type voltage-gated calcium channel, Cav1.2 alpha1 subunit. (Q30765972) (← links)
- Selective modulation of cellular voltage-dependent calcium channels by hyperbaric pressure-a suggested HPNS partial mechanism (Q33666310) (← links)
- Molecular determinants of inactivation in voltage-gated Ca2 channels (Q34061997) (← links)
- Micro-processing events in mRNAs identified by DHPLC analysis (Q34367120) (← links)
- Functional diversity in neuronal voltage-gated calcium channels by alternative splicing of Ca(v)alpha1. (Q34976636) (← links)
- The impact of splice isoforms on voltage-gated calcium channel alpha1 subunits (Q35596481) (← links)
- Novel splice variants of rat CaV2.1 that lack much of the synaptic protein interaction site are expressed in neuroendocrine cells (Q35676370) (← links)
- The active-zone protein Munc13 controls the use-dependence of presynaptic voltage-gated calcium channels (Q35917973) (← links)
- A hot spot for the interaction of gating modifier toxins with voltage-dependent ion channels (Q36436309) (← links)
- Interactions among toxins that inhibit N-type and P-type calcium channels (Q36558159) (← links)
- Selective pressure modulation of synaptic voltage-dependent calcium channels-involvement in HPNS mechanism. (Q37251568) (← links)
- Linker flexibility of IVS3-S4 loops modulates voltage-dependent activation of L-type Ca2 channels. (Q37613802) (← links)
- The neuronal splicing factor Nova controls alternative splicing in N-type and P-type CaV2 calcium channels (Q37819803) (← links)
- P/Q-type calcium channel modulators (Q38016449) (← links)
- Characterization of the SubstitutedN-Triazole Oxindole TROX-1, a Small-Molecule, State-Dependent Inhibitor of Cav2 Calcium Channels (Q39424475) (← links)
- Developmental control of CaV1.2 L-type calcium channel splicing by Fox proteins (Q39831315) (← links)
- The calcium channel alpha2delta-2 subunit partitions with CaV2.1 into lipid rafts in cerebellum: implications for localization and function. (Q40240262) (← links)
- Alternative splicing as a molecular switch for Ca2 /calmodulin-dependent facilitation of P/Q-type Ca2 channels. (Q40535862) (← links)
- Alternative splicing in the voltage-sensing region of N-Type CaV2.2 channels modulates channel kinetics (Q40544608) (← links)
- Functional consequences of P/Q-type Ca2 channel Cav2.1 missense mutations associated with episodic ataxia type 2 and progressive ataxia (Q40763673) (← links)
- Expression and characterization of recombinant kurtoxin, an inhibitor of T-type voltage-gated calcium channels (Q43405596) (← links)
- The ducky mutation in Cacna2d2 results in altered Purkinje cell morphology and is associated with the expression of a truncated alpha 2 delta-2 protein with abnormal function (Q43833683) (← links)
- Alternative splicing of a beta4 subunit proline-rich motif regulates voltage-dependent gating and toxin block of Cav2.1 Ca2 channels. (Q44856248) (← links)
- C-Terminal alternative splicing changes the gating properties of a human spinal cord calcium channel alpha 1A subunit. (Q44874131) (← links)
- Dominant-negative calcium channel suppression by truncated constructs involves a kinase implicated in the unfolded protein response. (Q44931369) (← links)
- Dominant-negative effects of human P/Q-type Ca2 channel mutations associated with episodic ataxia type 2. (Q46818398) (← links)
- Synaptic calcium-channel function in Drosophila: analysis and transformation rescue of temperature-sensitive paralytic and lethal mutations of cacophony. (Q47071378) (← links)
- Alternative splicing of a short cassette exon in alpha1B generates functionally distinct N-type calcium channels in central and peripheral neurons (Q47954183) (← links)
- Maturation of rat cerebellar Purkinje cells reveals an atypical Ca2 channel current that is inhibited by omega-agatoxin IVA and the dihydropyridine (-)-(S)-Bay K8644. (Q48359638) (← links)
- Alternative splicing generates a smaller assortment of CaV2.1 transcripts in cerebellar Purkinje cells than in the cerebellum. (Q48702276) (← links)
- Lambert-Eaton syndrome antibodies target multiple subunits of voltage-gated Ca2 channels (Q50455790) (← links)
- Amino acids in segment IVS6 and beta-subunit interaction support distinct conformational changes during Ca(v)2.1 inactivation (Q50715590) (← links)
- A novel extracellular calcium sensing mechanism in voltage-gated potassium ion channels (Q74015223) (← links)
- Regulation of signal transduction in Coilia nasus during migration (Q92567538) (← links)