Pages that link to "Q24522411"
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The following pages link to Clostridium difficile toxin B induces apoptosis in intestinal cultured cells (Q24522411):
Displaying 36 items.
- Clostridium difficile toxins: mechanism of action and role in disease (Q24522454) (← links)
- Blastocystis ratti induces contact-independent apoptosis, F-actin rearrangement, and barrier function disruption in IEC-6 cells (Q24549436) (← links)
- Rho activation patterns after spinal cord injury and the role of activated Rho in apoptosis in the central nervous system (Q24675542) (← links)
- Clostridium difficile Toxins A and B: Insights into Pathogenic Properties and Extraintestinal Effects (Q26751477) (← links)
- Coral pathogens identified for White Syndrome (WS) epizootics in the Indo-Pacific (Q27301085) (← links)
- Clostridium difficile Toxin B causes epithelial cell necrosis through an autoprocessing-independent mechanism (Q27339318) (← links)
- The A3 adenosine receptor induces cytoskeleton rearrangement in human astrocytoma cells via a specific action on Rho proteins (Q28344385) (← links)
- Intestinal epithelial restitution after TcdB challenge and recovery from Clostridium difficile infection in mice with alanyl-glutamine treatment (Q30413086) (← links)
- Systems analysis of the transcriptional response of human ileocecal epithelial cells to Clostridium difficile toxins and effects on cell cycle control (Q30428655) (← links)
- Clostridium difficile toxin B is an inflammatory enterotoxin in human intestine. (Q30973103) (← links)
- Strain-dependent induction of enterocyte apoptosis by Giardia lamblia disrupts epithelial barrier function in a caspase-3-dependent manner (Q31078416) (← links)
- Strain-dependent induction of epithelial cell oncosis by Campylobacter jejuni is correlated with invasion ability and is independent of cytolethal distending toxin (Q33910290) (← links)
- Vibrio parahaemolyticus thermostable direct hemolysin modulates cytoskeletal organization and calcium homeostasis in intestinal cultured cells (Q34000552) (← links)
- The interplay between the Escherichia coli Rho guanine nucleotide exchange factor effectors and the mammalian RhoGEF inhibitor EspH (Q34247723) (← links)
- Hijacking Rho GTPases by protein toxins and apoptosis: molecular strategies of pathogenic bacteria. (Q35109696) (← links)
- Colonic IgA producing cells and macrophages are reduced in recurrent and non-recurrent Clostridium difficile associated diarrhoea (Q35587936) (← links)
- Proteinase-activated receptor 1 activation induces epithelial apoptosis and increases intestinal permeability (Q35979002) (← links)
- Clostridium difficile Toxin A Undergoes Clathrin-Independent, PACSIN2-Dependent Endocytosis (Q36220118) (← links)
- Sustained polymorphonuclear leukocyte transmigration induces apoptosis in T84 intestinal epithelial cells (Q36293736) (← links)
- The intestinal microbiota dysbiosis and Clostridium difficile infection: is there a relationship with inflammatory bowel disease? (Q36515597) (← links)
- Regulation of Apoptosis by Gram-Positive Bacteria: Mechanistic Diversity and Consequences for Immunity (Q37009989) (← links)
- Functional implications of lethal toxin-catalysed glucosylation of (H/K/N)Ras and Rac1 in Clostridium sordellii-associated disease (Q37816677) (← links)
- The enterotoxicity of Clostridium difficile toxins (Q37954308) (← links)
- Enteric glial cells are susceptible to Clostridium difficile toxin B. (Q38728643) (← links)
- Clostridium difficile toxin A attenuates Wnt/β-catenin signaling in intestinal epithelial cells (Q39006646) (← links)
- Glucosyltransferase Activity of Clostridium difficile Toxin B Triggers Autophagy-mediated Cell Growth Arrest. (Q40065545) (← links)
- Difference in the cytotoxic effects of toxin B from Clostridium difficile strain VPI 10463 and toxin B from variant Clostridium difficile strain 1470. (Q40200671) (← links)
- BNIP-Salpha induces cell rounding and apoptosis by displacing p50RhoGAP and facilitating RhoA activation via its unique motifs in the BNIP-2 and Cdc42GAP homology domain. (Q40343243) (← links)
- High Mobility Group Box1 Protein Is Involved in Endoplasmic Reticulum Stress Induced by Clostridium difficile Toxin A. (Q40556940) (← links)
- Lethal toxin fromClostridium sordelliiinduces apoptotic cell death by disruption of mitochondrial homeostasis in HL-60 cells (Q40628289) (← links)
- Clostridium difficile Toxins TcdA and TcdB Cause Colonic Tissue Damage by Distinct Mechanisms (Q41193374) (← links)
- Effects of Clostridium difficile toxin A and B on human T lymphocyte migration (Q41811642) (← links)
- Difference in the biological effects of Clostridium difficile toxin B in proliferating and non-proliferating cells (Q42816117) (← links)
- The role of toxins in Clostridium difficile infection. (Q47734724) (← links)
- N -Acetylcysteine protects epithelial cells against the oxidative imbalance due to Clostridium difficile toxins (Q60687752) (← links)
- Purification and evaluation of large clostridial cytotoxins that inhibit small GTPases of Rho and Ras subfamilies (Q73089636) (← links)