Pages that link to "Q24321418"
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The following pages link to CCDC98 targets BRCA1 to DNA damage sites (Q24321418):
Displaying 50 items.
- BRCA1 DNA repair associated (Q17487737) (← links)
- Abraxas 1, BRCA1 A complex subunit (Q21100476) (← links)
- Ubiquitin interaction motif containing 1 (Q21100482) (← links)
- PALB2 is an integral component of the BRCA complex required for homologous recombination repair (Q24316113) (← links)
- MERIT40 controls BRCA1-Rap80 complex integrity and recruitment to DNA double-strand breaks (Q24317241) (← links)
- MERIT40 facilitates BRCA1 localization and DNA damage repair (Q24317291) (← links)
- Molecular insights into the function of RING finger (RNF)-containing proteins hRNF8 and hRNF168 in Ubc13/Mms2-dependent ubiquitylation (Q24633548) (← links)
- BRCT domains: easy as one, two, three (Q24635835) (← links)
- Role of Deubiquitinating Enzymes in DNA Repair (Q26773800) (← links)
- Protein degradation and the stress response (Q26823693) (← links)
- Phosphopeptide interactions with BRCA1 BRCT domains: More than just a motif (Q26999072) (← links)
- Molecular basis of BACH1/FANCJ recognition by TopBP1 in DNA replication checkpoint control (Q27666163) (← links)
- Histone ubiquitination associates with BRCA1-dependent DNA damage response (Q28506770) (← links)
- Homologs of breast cancer genes in plants (Q28729894) (← links)
- Dual-fluorophore quantitative high-throughput screen for inhibitors of BRCT-phosphoprotein interaction (Q33312344) (← links)
- Mutation screening of the MERIT40 gene encoding a novel BRCA1 and RAP80 interacting protein in breast cancer families (Q33824592) (← links)
- Long-range massively parallel mate pair sequencing detects distinct mutations and similar patterns of structural mutability in two breast cancer cell lines (Q34036642) (← links)
- The BRCA1-interacting protein Abraxas is required for genomic stability and tumor suppression. (Q34109150) (← links)
- BRCA1 and BRCA2: breast/ovarian cancer susceptibility gene products and participants in DNA double-strand break repair (Q34110729) (← links)
- Association of BRCA1 and BRCA2 mutations with survival, chemotherapy sensitivity, and gene mutator phenotype in patients with ovarian cancer (Q34155131) (← links)
- BRCA1-directed, enhanced and aberrant homologous recombination: mechanism and potential treatment strategies (Q34252163) (← links)
- Loss of BRCA1-A complex function in RAP80 null tumor cells (Q34336205) (← links)
- Mutation of the BRCA1 SQ-cluster results in aberrant mitosis, reduced homologous recombination, and a compensatory increase in non-homologous end joining (Q34491545) (← links)
- BRCA1, PARP, and 53BP1: conditional synthetic lethality and synthetic viability (Q34539782) (← links)
- Structural maintenance of chromosomes flexible hinge domain containing 1 (SMCHD1) promotes non-homologous end joining and inhibits homologous recombination repair upon DNA damage (Q34634191) (← links)
- PARP1-driven poly-ADP-ribosylation regulates BRCA1 function in homologous recombination-mediated DNA repair. (Q34644116) (← links)
- RAP80-directed tuning of BRCA1 homologous recombination function at ionizing radiation-induced nuclear foci (Q34762756) (← links)
- The BRCA1-RAP80 complex regulates DNA repair mechanism utilization by restricting end resection (Q34787446) (← links)
- The role of BRCA1 in DNA damage response (Q34807044) (← links)
- RNF8-dependent histone ubiquitination during DNA damage response and spermatogenesis (Q34829400) (← links)
- Human Wrnip1 is localized in replication factories in a ubiquitin-binding zinc finger-dependent manner (Q35676889) (← links)
- BRCA1 tumor suppressor network: focusing on its tail (Q35861690) (← links)
- Irreversible binding of an anticancer compound (BI-94) to plasma proteins (Q36005906) (← links)
- ABRAXAS (FAM175A) and Breast Cancer Susceptibility: No Evidence of Association in the Breast Cancer Family Registry (Q36043159) (← links)
- RAP80 protein is important for genomic stability and is required for stabilizing BRCA1-A complex at DNA damage sites in vivo (Q36080420) (← links)
- Rap80 protein recruitment to DNA double-strand breaks requires binding to both small ubiquitin-like modifier (SUMO) and ubiquitin conjugates (Q36127031) (← links)
- BRCA1 Is Required for Maintenance of Phospho-Chk1 and G2/M Arrest during DNA Cross-Link Repair in DT40 Cells (Q36174109) (← links)
- RAP80 is critical in maintaining genomic stability and suppressing tumor development (Q36352398) (← links)
- TRAIP/RNF206 is required for recruitment of RAP80 to sites of DNA damage (Q36528092) (← links)
- Cdk1 protein-mediated phosphorylation of receptor-associated protein 80 (RAP80) serine 677 modulates DNA damage-induced G2/M checkpoint and cell survival (Q36596128) (← links)
- MDC1 and RNF8 function in a pathway that directs BRCA1-dependent localization of PALB2 required for homologous recombination (Q36648222) (← links)
- Ubc13/Rnf8 ubiquitin ligases control foci formation of the Rap80/Abraxas/Brca1/Brcc36 complex in response to DNA damage (Q36696336) (← links)
- RAP80 regulates epithelial-mesenchymal transition related with metastasis and malignancy of cancer (Q36746310) (← links)
- The interaction between CtIP and BRCA1 is not essential for resection-mediated DNA repair or tumor suppression (Q36878037) (← links)
- Kinetic analysis of interaction of BRCA1 tandem breast cancer c-terminal domains with phosphorylated peptides reveals two binding conformations (Q36952019) (← links)
- Genetic evaluation of BRCA1 associated a complex genes with triple-negative breast cancer susceptibility in Chinese women (Q36962780) (← links)
- Focus on histone variant H2AX: to be or not to be. (Q36963162) (← links)
- RNF8 promotes epithelial-mesenchymal transition of breast cancer cells (Q36969257) (← links)
- The DNA damage response pathways: at the crossroad of protein modifications (Q37034923) (← links)
- Recognition of DNA double strand breaks by the BRCA1 tumor suppressor network (Q37120275) (← links)