Pages that link to "Q24316341"
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The following pages link to Additional sex comb-like 1 (ASXL1), in cooperation with SRC-1, acts as a ligand-dependent coactivator for retinoic acid receptor (Q24316341):
Displaying 50 items.
- retinoic acid receptor alpha (Q254943) (← links)
- Nuclear receptor subfamily 3 group C member 1 (Q2663095) (← links)
- Nuclear receptor coactivator 1 (Q3681842) (← links)
- estrogen receptor 1 (Q5401858) (← links)
- Nuclear receptor corepressor 1 (Q7068191) (← links)
- ASXL transcriptional regulator 1 (Q21116525) (← links)
- Retinoid X receptor alpha (Q21499097) (← links)
- ASXL transcriptional regulator 1 (Q21499098) (← links)
- ASXL1 mutations promote myeloid transformation through loss of PRC2-mediated gene repression (Q24296658) (← links)
- Negative regulation of ERα by a novel protein CAC1 through association with histone demethylase LSD1 (Q24304751) (← links)
- Additional Sex Comb-like (ASXL) Proteins 1 and 2 Play Opposite Roles in Adipogenesis via Reciprocal Regulation of Peroxisome Proliferator-activated Receptor γ (Q24305362) (← links)
- MED25 is distinct from TRAP220/MED1 in cooperating with CBP for retinoid receptor activation (Q24321381) (← links)
- The ubiquitin carboxyl hydrolase BAP1 forms a ternary complex with YY1 and HCF-1 and is a critical regulator of gene expression (Q24633127) (← links)
- ASXL1 mutations identify a high-risk subgroup of older patients with primary cytogenetically normal AML within the ELN Favorable genetic category (Q27851679) (← links)
- ASXL1 exon 12 mutations are frequent in AML with intermediate risk karyotype and are independently associated with an adverse outcome (Q27851909) (← links)
- ASXL1 represses retinoic acid receptor-mediated transcription through associating with HP1 and LSD1 (Q28115338) (← links)
- Loss-of-function Additional sex combs like 1 mutations disrupt hematopoiesis but do not cause severe myelodysplasia or leukemia (Q28585869) (← links)
- Myelodysplastic syndromes are induced by histone methylation–altering ASXL1 mutations (Q28590040) (← links)
- Kinase activity-independent suppression of p73alpha by AMP-activated kinase alpha (AMPKalpha). (Q33392580) (← links)
- Additional sex combs-like 1 belongs to the enhancer of trithorax and polycomb group and genetically interacts with Cbx2 in mice (Q33595603) (← links)
- The HARE-HTH and associated domains: novel modules in the coordination of epigenetic DNA and protein modifications (Q34107107) (← links)
- Piperine, a Component of Black Pepper, Inhibits Adipogenesis by Antagonizing PPARγ Activity in 3T3-L1 Cells (Q34264798) (← links)
- Current outlook on molecular pathogenesis and treatment of myeloproliferative neoplasms (Q34302770) (← links)
- Functional proteomics of the epigenetic regulators ASXL1, ASXL2 and ASXL3: a convergence of proteomics and epigenetics for translational medicine (Q34469971) (← links)
- The BAP1/ASXL2 Histone H2A Deubiquitinase Complex Regulates Cell Proliferation and Is Disrupted in Cancer (Q34496129) (← links)
- Mutations in epigenetic modifiers in the pathogenesis and therapy of acute myeloid leukemia. (Q34651312) (← links)
- Additional sex combs-like 2 is required for polycomb repressive complex 2 binding at select targets (Q34989584) (← links)
- The molecular basis of myeloid malignancies (Q35103708) (← links)
- De novo dominant ASXL3 mutations alter H2A deubiquitination and transcription in Bainbridge-Ropers syndrome. (Q36514637) (← links)
- Isolation and characterization of a novel H1.2 complex that acts as a repressor of p53-mediated transcription. (Q36727277) (← links)
- Loss of Asxl1 Alters Self-Renewal and Cell Fate of Bone Marrow Stromal Cell, Leading to Bohring-Opitz-like Syndrome in Mice (Q37014700) (← links)
- Functional and cancer genomics of ASXL family members (Q37040682) (← links)
- ASXL1 plays an important role in erythropoiesis (Q37049843) (← links)
- Heterogeneous breakpoints in patients with acute lymphoblastic leukemia and the dic(9;20)(p11-13;q11) show recurrent involvement of genes at 20q11.21. (Q37284420) (← links)
- Loss of Asxl1 leads to myelodysplastic syndrome-like disease in mice (Q37512080) (← links)
- Unraveling the Molecular Pathophysiology of Myelodysplastic Syndromes (Q37671806) (← links)
- Molecular aspects of myeloproliferative neoplasms. (Q37699001) (← links)
- JAK-Mutant Myeloproliferative Neoplasms (Q37913220) (← links)
- The pathogenesis of essential thrombocythemia (Q37913902) (← links)
- Update on cytogenetic and molecular changes in myelodysplastic syndromes (Q37923939) (← links)
- Molecular biology of myelodysplastic syndromes (Q37937809) (← links)
- Mutations in epigenetic regulators in myelodysplastic syndromes (Q37974844) (← links)
- Mutations in ASXL1 are associated with poor prognosis across the spectrum of malignant myeloid diseases (Q37995804) (← links)
- Role of TET2 and ASXL1 mutations in the pathogenesis of myeloproliferative neoplasms (Q38046319) (← links)
- The ASXL-BAP1 axis: new factors in myelopoiesis, cancer and epigenetics (Q38059391) (← links)
- The genetic basis of phenotypic heterogeneity in myelodysplastic syndromes (Q38061848) (← links)
- Genetic and epigenetic alterations of myeloproliferative disorders (Q38066786) (← links)
- Genetic basis of MPN: Beyond JAK2-V617F. (Q38159973) (← links)
- Asxl1 deficiency in embryonic fibroblasts leads to cellular senescence via impairment of the AKT-E2F pathway and Ezh2 inactivation (Q38401146) (← links)
- ASXL1 interacts with the cohesin complex to maintain chromatid separation and gene expression for normal hematopoiesis (Q39003064) (← links)