Pages that link to "Q24300673"

The following pages link to Identification and validation of the mitochondrial F1F0-ATPase as the molecular target of the immunomodulatory benzodiazepine Bz-423 (Q24300673):

Displaying 49 items.

• ATP synthase peripheral stalk subunit OSCP (Q21109360) (← links)
• Redox-directed cancer therapeutics: molecular mechanisms and opportunities (Q24645584) (← links)
• The Dual Function of Reactive Oxygen/Nitrogen Species in Bioenergetics and Cell Death: The Role of ATP Synthase (Q26749074) (← links)
• From ATP to PTP and Back: A Dual Function for the Mitochondrial ATP Synthase (Q27004110) (← links)
• Mechanism of inhibition of bovine F1-ATPase by resveratrol and related polyphenols (Q27647277) (← links)
• The mitochondrial permeability transition pore: molecular nature and role as a target in cardioprotection (Q28248925) (← links)
• Dimers of mitochondrial ATP synthase form the permeability transition pore (Q28287824) (← links)
• Identification of gp96 as a novel target for treatment of autoimmune disease in mice (Q28473392) (← links)
• Regulation of the mitochondrial permeability transition pore by the outer membrane does not involve the peripheral benzodiazepine receptor (Translocator Protein of 18 kDa (TSPO)). (Q33619335) (← links)
• Use of phage display technology for the determination of the targets for small-molecule therapeutics (Q34347887) (← links)
• Anaplerotic metabolism of alloreactive T cells provides a metabolic approach to treat graft-versus-host disease. (Q34360724) (← links)
• F-ATPase of Drosophila melanogaster forms 53-picosiemen (53-pS) channels responsible for mitochondrial Ca2 -induced Ca2 release (Q35103850) (← links)
• Tyrosine kinase inhibition in leukemia induces an altered metabolic state sensitive to mitochondrial perturbations (Q35178420) (← links)
• Thymoquinone Inhibits Escherichia coli ATP Synthase and Cell Growth (Q35634052) (← links)
• Manipulating the bioenergetics of alloreactive T cells causes their selective apoptosis and arrests graft-versus-host disease (Q35998783) (← links)
• The Mitochondrial Permeability Transition Pore: Channel Formation by F-ATP Synthase, Integration in Signal Transduction, and Role in Pathophysiology. (Q36146320) (← links)
• Distinct metabolic programs in activated T cells: opportunities for selective immunomodulation (Q36174985) (← links)
• Matched and mismatched metabolic fuels in lymphocyte function (Q36640334) (← links)
• Metabolic regulation of T lymphocytes (Q36711431) (← links)
• ATP synthase and the actions of inhibitors utilized to study its roles in human health, disease, and other scientific areas (Q36995302) (← links)
• Apoptotic signaling activated by modulation of the F0F1-ATPase: implications for selective killing of autoimmune lymphocytes (Q37416439) (← links)
• Structure of the mitochondrial ATP synthase from Pichia angusta determined by electron cryo-microscopy (Q37417752) (← links)
• Translocator Protein 18 kDa (TSPO): An Old Protein with New Functions? (Q37444435) (← links)
• The metabolic life and times of a T-cell (Q37773430) (← links)
• The mitochondrial permeability transition pore: a mystery solved? (Q38106976) (← links)
• Hitting the Bull's-Eye in Metastatic Cancers-NSAIDs Elevate ROS in Mitochondria, Inducing Malignant Cell Death. (Q38358175) (← links)
• Mitochondrial Ca2 and regulation of the permeability transition pore (Q38920740) (← links)
• In vivo inhibition of the mitochondrial H -ATP synthase in neurons promotes metabolic preconditioning. (Q38972904) (← links)
• Fine tuning of immunometabolism for the treatment of rheumatic diseases (Q39223057) (← links)
• Mitochondrial control of immunity: beyond ATP. (Q39410087) (← links)
• Bz-423 superoxide signals apoptosis via selective activation of JNK, Bak, and Bax (Q39579957) (← links)
• Bz-423 superoxide signals B cell apoptosis via Mcl-1, Bak, and Bax. (Q39845306) (← links)
• A therapeutic connection between dietary phytochemicals and ATP synthase (Q40083367) (← links)
• The oligomycin-sensitivity conferring protein of mitochondrial ATP synthase: emerging new roles in mitochondrial pathophysiology (Q40709485) (← links)
• Characterization of the respiration-induced yeast mitochondrial permeability transition pore (Q41945786) (← links)
• Mechanistic basis for differential inhibition of the F1Fo-ATPase by aurovertin (Q42576659) (← links)
• The unique histidine in OSCP subunit of F-ATP synthase mediates inhibition of the permeability transition pore by acidic pH. (Q47317776) (← links)
• Changes in Nutritional Status Impact Immune Cell Metabolism and Function. (Q55262568) (← links)
• Visualizing Mitochondrial FoF1-ATP Synthase as the Target of the Immunomodulatory Drug Bz-423. (Q55712250) (← links)
• OSCP subunit of mitochondrial ATP synthase: Role in regulation of enzyme function and of its transition to a pore (Q57020330) (← links)
• Mitochondrial F-ATP Synthase and Its Transition into an Energy-Dissipating Molecular Machine. (Q64986174) (← links)
• Purified F-ATP synthase forms a Ca2 -dependent high-conductance channel matching the mitochondrial permeability transition pore (Q90287798) (← links)
• A mitochondrial megachannel resides in monomeric F1FO ATP synthase (Q92162792) (← links)
• Targeting metabolism to regulate immune responses in autoimmunity and cancer (Q92297313) (← links)
• T cell metabolism: new insights in systemic lupus erythematosus pathogenesis and therapy (Q92712487) (← links)
• Molecular nature and regulation of the mitochondrial permeability transition pore(s), drug target(s) in cardioprotection (Q95848133) (← links)
• ATP synthase c-subunit ring as the channel of mitochondrial permeability transition: Regulator of metabolism in development and degeneration (Q95930009) (← links)
• Mitochondrial calcium signalling and neurodegenerative diseases (Q97884849) (← links)
• Natural products and other inhibitors of F1FO ATP synthase (Q99562226) (← links)