Pages that link to "Q18195722"
Jump to navigation
Jump to search
The following pages link to David Ron (Q18195722):
Displaying 50 items.
- Coupling of stress in the ER to activation of JNK protein kinases by transmembrane protein kinase IRE1 (Q22011167) (← links)
- A membrane protein complex mediates retro-translocation from the ER lumen into the cytosol (Q24297732) (← links)
- Disulphide production by Ero1α-PDI relay is rapid and effectively regulated (Q24297912) (← links)
- Fusion of CHOP to a novel RNA-binding protein in human myxoid liposarcoma (Q24309359) (← links)
- Heightened stress response in primary fibroblasts expressing mutant eIF2B genes from CACH/VWM leukodystrophy patients (Q24309616) (← links)
- Heat shock protein 90 modulates the unfolded protein response by stabilizing IRE1alpha (Q24320638) (← links)
- Regulated association of misfolded endoplasmic reticulum lumenal proteins with P58/DNAJc3 (Q24324029) (← links)
- Identification of novel stress-induced genes downstream of chop (Q24533261) (← links)
- Control of PERK eIF2alpha kinase activity by the endoplasmic reticulum stress-induced molecular chaperone P58IPK. (Q24541469) (← links)
- Somatic CALR mutations in myeloproliferative neoplasms with nonmutated JAK2 (Q24568313) (← links)
- The molecular basis for selective inhibition of unconventional mRNA splicing by an IRE1-binding small molecule (Q24596804) (← links)
- The gadd and MyD genes define a novel set of mammalian genes encoding acidic proteins that synergistically suppress cell growth (Q24613548) (← links)
- Transmission of cell stress from endoplasmic reticulum to mitochondria: enhanced expression of Lon protease (Q24672509) (← links)
- Divergent effects of PERK and IRE1 signaling on cell viability (Q27438124) (← links)
- Crystal Structure of P58(IPK) TPR Fragment Reveals the Mechanism for its Molecular Chaperone Activity in UPR (Q27660049) (← links)
- Flavonol Activation Defines an Unanticipated Ligand-Binding Site in the Kinase-RNase Domain of IRE1 (Q27660827) (← links)
- The structure of the PERK kinase domain suggests the mechanism for its activation (Q27667715) (← links)
- Signal integration in the endoplasmic reticulum unfolded protein response (Q27860577) (← links)
- Human 75-kDa DNA-pairing protein is identical to the pro-oncoprotein TLS/FUS and is able to promote D-loop formation (Q28138179) (← links)
- Perk is essential for translational regulation and cell survival during the unfolded protein response (Q28140062) (← links)
- Rearrangement of the transcription factor gene CHOP in myxoid liposarcomas with t(12;16)(q13;p11) (Q28183334) (← links)
- IRE1 couples endoplasmic reticulum load to secretory capacity by processing the XBP-1 mRNA (Q28214814) (← links)
- A novel effector domain from the RNA-binding protein TLS or EWS is required for oncogenic transformation by CHOP (Q28240554) (← links)
- Characterization of the CHOP breakpoints and fusion transcripts in myxoid liposarcomas with the 12;16 translocation (Q28242489) (← links)
- TLS (FUS) binds RNA in vivo and engages in nucleo-cytoplasmic shuttling (Q28246694) (← links)
- TLS (translocated-in-liposarcoma) is a high-affinity interactor for steroid, thyroid hormone, and retinoid receptors (Q28259754) (← links)
- Stress-induced phosphorylation and activation of the transcription factor CHOP (GADD153) by p38 MAP Kinase (Q28280649) (← links)
- CHOP, a novel developmentally regulated nuclear protein that dimerizes with transcription factors C/EBP and LAP and functions as a dominant-negative inhibitor of gene transcription (Q28287446) (← links)
- Protein translation and folding are coupled by an endoplasmic-reticulum-resident kinase (Q28296183) (← links)
- Expression patterns of the human sarcoma-associated genes FUS and EWS and the genomic structure of FUS (Q28296815) (← links)
- A selective inhibitor of eIF2alpha dephosphorylation protects cells from ER stress (Q28306995) (← links)
- ERAD inhibitors integrate ER stress with an epigenetic mechanism to activate BH3-only protein NOXA in cancer cells (Q28307506) (← links)
- Arsenite-inducible RNA-associated protein (AIRAP) protects cells from arsenite toxicity (Q28347031) (← links)
- Increased sensitivity to dextran sodium sulfate colitis in IRE1beta-deficient mice (Q28359871) (← links)
- Hyperhomocysteinemia and function of the endoplasmic reticulum (Q28365879) (← links)
- Modulation of Innate Immune Signalling by Lipid-Mediated MAVS Transmembrane Domain Oligomerization (Q28387082) (← links)
- Mammalian stress granules represent sites of accumulation of stalled translation initiation complexes (Q28504910) (← links)
- Cotranslocational degradation protects the stressed endoplasmic reticulum from protein overload (Q28506232) (← links)
- Regulated translation initiation controls stress-induced gene expression in mammalian cells (Q28506388) (← links)
- Activating transcription factor 3 is integral to the eukaryotic initiation factor 2 kinase stress response (Q28508156) (← links)
- Endoplasmic reticulum thiol oxidase deficiency leads to ascorbic acid depletion and noncanonical scurvy in mice (Q28508619) (← links)
- CHOP is implicated in programmed cell death in response to impaired function of the endoplasmic reticulum (Q28509611) (← links)
- The GCN2 kinase biases feeding behavior to maintain amino acid homeostasis in omnivores (Q28511717) (← links)
- Feedback inhibition of the unfolded protein response by GADD34-mediated dephosphorylation of eIF2alpha (Q28512201) (← links)
- CHOP induces death by promoting protein synthesis and oxidation in the stressed endoplasmic reticulum (Q28512249) (← links)
- Inhibition of a constitutive translation initiation factor 2alpha phosphatase, CReP, promotes survival of stressed cells (Q28513964) (← links)
- Stress-induced binding of the transcriptional factor CHOP to a novel DNA control element (Q28570085) (← links)
- CHOP/GADD153 is a mediator of apoptotic death in substantia nigra dopamine neurons in an in vivo neurotoxin model of parkinsonism (Q28579022) (← links)
- Antiviral effect of the mammalian translation initiation factor 2alpha kinase GCN2 against RNA viruses (Q28586515) (← links)
- Role of ERO1-alpha-mediated stimulation of inositol 1,4,5-triphosphate receptor activity in endoplasmic reticulum stress-induced apoptosis (Q28586997) (← links)