Pages that link to "Q16199016"

The following pages link to Paolo Bernardi (Q16199016):

Displaying 50 items.

• Paolo Bernardi (Q3893768) (← links)
• Antamanide, a derivative of Amanita phalloides, is a novel inhibitor of the mitochondrial permeability transition pore (Q21562115) (← links)
• The mitochondrial chaperone TRAP1 promotes neoplastic growth by inhibiting succinate dehydrogenase (Q24294745) (← links)
• Hexokinase II detachment from mitochondria triggers apoptosis through the permeability transition pore independent of voltage-dependent anion channels (Q24312702) (← links)
• Developmental shift of cyclophilin D contribution to hypoxic-ischemic brain injury (Q24607913) (← links)
• Cyclosporin A corrects mitochondrial dysfunction and muscle apoptosis in patients with collagen VI myopathies (Q24647603) (← links)
• From ATP to PTP and Back: A Dual Function for the Mitochondrial ATP Synthase (Q27004110) (← links)
• The mitochondrial permeability transition pore: molecular nature and role as a target in cardioprotection (Q28248925) (← links)
• The mitochondrial permeability transition from yeast to mammals (Q28279895) (← links)
• Dimers of mitochondrial ATP synthase form the permeability transition pore (Q28287824) (← links)
• Regulation of the inner membrane mitochondrial permeability transition by the outer membrane translocator protein (peripheral benzodiazepine receptor) (Q28297948) (← links)
• Cyclophilin D modulates mitochondrial F0F1-ATP synthase by interacting with the lateral stalk of the complex (Q28507223) (← links)
• Phosphate is essential for inhibition of the mitochondrial permeability transition pore by cyclosporin A and by cyclophilin D ablation (Q28863159) (← links)
• Myotonic dystrophy protein kinase (DMPK) prevents ROS-induced cell death by assembling a hexokinase II-Src complex on the mitochondrial surface (Q30009539) (← links)
• Ligand-selective modulation of the permeability transition pore by arginine modification. Opposing effects of p-hydroxyphenylglyoxal and phenylglyoxal. (Q31028811) (← links)
• Opening of the mitochondrial permeability transition pore causes depletion of mitochondrial and cytosolic NAD and is a causative event in the death of myocytes in postischemic reperfusion of the heart. (Q31552541) (← links)
• The mitochondrial permeability transition, release of cytochrome c and cell death. Correlation with the duration of pore openings in situ (Q31779663) (← links)
• Modification of permeability transition pore arginine(s) by phenylglyoxal derivatives in isolated mitochondria and mammalian cells. Structure-function relationship of arginine ligands. (Q33211203) (← links)
• The mitochondrial effects of small organic ligands of BCL-2: sensitization of BCL-2-overexpressing cells to apoptosis by a pyrimidine-2,4,6-trione derivative (Q33234059) (← links)
• High concordance of drug-induced human hepatotoxicity with in vitro cytotoxicity measured in a novel cell-based model using high content screening (Q33239100) (← links)
• Properties of the permeability transition in VDAC1(-/-) mitochondria (Q33240486) (← links)
• Regulation of the mitochondrial permeability transition pore by the outer membrane does not involve the peripheral benzodiazepine receptor (Translocator Protein of 18 kDa (TSPO)). (Q33619335) (← links)
• Channel formation by yeast F-ATP synthase and the role of dimerization in the mitochondrial permeability transition (Q33718426) (← links)
• Mitochondria and cell death. Mechanistic aspects and methodological issues (Q33734610) (← links)
• Mitochondrial transport of cations: channels, exchangers, and permeability transition. (Q33744633) (← links)
• Progress on the mitochondrial permeability transition pore: regulation by complex I and ubiquinone analogs. (Q33832698) (← links)
• The effects of idebenone on mitochondrial bioenergetics (Q34075134) (← links)
• Destabilization of the outer and inner mitochondrial membranes by core and linker histones (Q34241828) (← links)
• Monoamine oxidase inhibition prevents mitochondrial dysfunction and apoptosis in myoblasts from patients with collagen VI myopathies (Q34266732) (← links)
• Mitochondrial dysfunction and defective autophagy in the pathogenesis of collagen VI muscular dystrophies. (Q34338854) (← links)
• p66Shc, mitochondria, and the generation of reactive oxygen species. (Q34357221) (← links)
• Properties of the permeability transition pore in mitochondria devoid of Cyclophilin D. (Q34406704) (← links)
• Melanocytes from Patients Affected by Ullrich Congenital Muscular Dystrophy and Bethlem Myopathy have Dysfunctional Mitochondria That Can be Rescued with Cyclophilin Inhibitors (Q34547398) (← links)
• Changes in muscle cell metabolism and mechanotransduction are associated with myopathic phenotype in a mouse model of collagen VI deficiency. (Q34599342) (← links)
• Assessing the molecular basis for rat-selective induction of the mitochondrial permeability transition by norbormide (Q34629293) (← links)
• Enhancement of anxiety, facilitation of avoidance behavior, and occurrence of adult-onset obesity in mice lacking mitochondrial cyclophilin D (Q34671714) (← links)
• Therapy of collagen VI-related myopathies (Bethlem and Ullrich). (Q34883830) (← links)
• Peptide-based carbon nanotubes for mitochondrial targeting (Q34884970) (← links)
• Mitochondrial energy dissipation by fatty acids. Mechanisms and implications for cell death (Q35025248) (← links)
• Hepatic progenitor cells express SerpinB3 (Q35092061) (← links)
• F-ATPase of Drosophila melanogaster forms 53-picosiemen (53-pS) channels responsible for mitochondrial Ca2 -induced Ca2 release (Q35103850) (← links)
• Mitochondria and reperfusion injury. The role of permeability transition (Q35166967) (← links)
• Mitochondrial deoxynucleotide pool sizes in mouse liver and evidence for a transport mechanism for thymidine monophosphate (Q35214674) (← links)
• Cyclosporine A in Ullrich congenital muscular dystrophy: long-term results (Q35418243) (← links)
• Mitochondrial dysfunction in the pathogenesis of Ullrich congenital muscular dystrophy and prospective therapy with cyclosporins (Q35612416) (← links)
• Cyclophilin D inactivation protects axons in experimental autoimmune encephalomyelitis, an animal model of multiple sclerosis (Q35762075) (← links)
• Early resistance to cell death and to onset of the mitochondrial permeability transition during hepatocarcinogenesis with 2-acetylaminofluorene (Q35811320) (← links)
• Properties of Ca(2 ) transport in mitochondria of Drosophila melanogaster (Q35842069) (← links)
• The Mitochondrial Permeability Transition Pore: Channel Formation by F-ATP Synthase, Integration in Signal Transduction, and Role in Pathophysiology. (Q36146320) (← links)
• Critical evaluation of the use of cell cultures for inclusion in clinical trials of patients affected by collagen VI myopathies (Q36153344) (← links)