Direct evidence for a causative role of FGF23 in the abnormal renal phosphate handling and vitamin D metabolism in rats with early-stage chronic kidney disease (Q85036109)

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scientific article published on 15 September 2010

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English	Direct evidence for a causative role of FGF23 in the abnormal renal phosphate handling and vitamin D metabolism in rats with early-stage chronic kidney disease	scientific article published on 15 September 2010

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:20844473 AND SRC:MED&resulttype=core&format=json

14 February 2020

Direct evidence for a causative role of FGF23 in the abnormal renal phosphate handling and vitamin D metabolism in rats with early-stage chronic kidney disease (English)

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14 February 2020

chronic renal insufficiency

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8

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14 February 2020

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Hisashi Hasegawa

1

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14 February 2020

Nobuo Nagano

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14 February 2020

Itaru Urakawa

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14 February 2020

Yuji Yamazaki

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14 February 2020

Kousuke Iijima

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14 February 2020

Toshiro Fujita

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14 February 2020

Takeyoshi Yamashita

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14 February 2020

Takashi Shimada

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14 February 2020

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15 September 2010

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14 February 2020

Kidney International

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14 February 2020

78

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14 February 2020

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14 February 2020

975-980

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14 February 2020

Secondary hyperparathyroidism in renal failure: the trade-off hypothesis revisited

1 reference

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7 January 2021

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Endocrine functions of bone in mineral metabolism regulation.

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Bone as an endocrine organ.

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Increased circulatory level of biologically active full-length FGF-23 in patients with hypophosphatemic rickets/osteomalacia

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Fibroblast growth factor 23 in oncogenic osteomalacia and X-linked hypophosphatemia

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Autosomal dominant hypophosphataemic rickets is associated with mutations in FGF23

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Loss of DMP1 causes rickets and osteomalacia and identifies a role for osteocytes in mineral metabolism.

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DMP1 mutations in autosomal recessive hypophosphatemia implicate a bone matrix protein in the regulation of phosphate homeostasis.

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Cloning and characterization of FGF23 as a causative factor of tumor-induced osteomalacia

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Circulating concentration of FGF-23 increases as renal function declines in patients with chronic kidney disease, but does not change in response to variation in phosphate intake in healthy volunteers

1 reference

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7 January 2021

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inferred from DOI database lookup

Serum FGF23 levels in normal and disordered phosphorus homeostasis

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7 January 2021

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FGF-23 in patients with end-stage renal disease on hemodialysis

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Circulating fibroblast growth factor 23 in patients with end-stage renal disease treated by peritoneal dialysis is intact and biologically active

1 reference

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7 January 2021

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Altered divalent ion metabolism in early renal failure: role of 1,25(OH)2D

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7 January 2021

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Possible involvement of circulating fibroblast growth factor 23 in the development of secondary hyperparathyroidism associated with renal insufficiency.

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Fibroblast growth factor-23 mitigates hyperphosphatemia but accentuates calcitriol deficiency in chronic kidney disease

1 reference

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7 January 2021

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Anti-FGF23 neutralizing antibodies show the physiological role and structural features of FGF23.

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Therapeutic effects of anti-FGF23 antibodies in hypophosphatemic rickets/osteomalacia

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Sevelamer hydrochloride, a phosphate binder, protects against deterioration of renal function in rats with progressive chronic renal insufficiency

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Role of hyperphosphatemia and 1,25-dihydroxyvitamin D in vascular calcification and mortality in fibroblastic growth factor 23 null mice

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Effect of manipulating serum phosphorus with phosphate binder on circulating PTH and FGF23 in renal failure rats

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Effect of dietary phosphorus on circulating concentrations of 1,25-dihydroxyvitamin D and immunoreactive parathyroid hormone in children with moderate renal insufficiency

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Transgenic mice overexpressing human fibroblast growth factor 23 (R176Q) delineate a putative role for parathyroid hormone in renal phosphate wasting disorders

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Fibroblast growth factor-23 regulates parathyroid hormone and 1alpha-hydroxylase expression in cultured bovine parathyroid cells

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Depressed expression of Klotho and FGF receptor 1 in hyperplastic parathyroid glands from uremic patients.

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