Novel insights into the distribution of cardiac HCN channels: An expression study in the mouse heart (Q84980492)

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13 February 2020

title

Novel insights into the distribution of cardiac HCN channels: an expression study in the mouse heart (English)

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13 February 2020

Novel insights into the distribution of cardiac HCN channels: An expression study in the mouse heart (English)

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https://api.crossref.org/works/10.1016/J.YJMCC.2011.09.005

13 December 2024

author name string

Stefan Herrmann

series ordinal

1

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13 February 2020

Beate Layh

series ordinal

2

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13 February 2020

Andreas Ludwig

series ordinal

3

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13 February 2020

language of work or name

English

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National Center for Biotechnology Information

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=21945247

13 December 2024

publication date

14 September 2011

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full work available at URL

13 February 2020

https://api.elsevier.com/content/article/PII:S0022282811003816?httpAccept=text/xml

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https://api.elsevier.com/content/article/PII:S0022282811003816?httpAccept=text/plain

13 December 2024

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https://api.crossref.org/works/10.1016/J.YJMCC.2011.09.005

Journal of Molecular and Cellular Cardiology

13 December 2024

published in

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13 February 2020

volume

51

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13 February 2020

issue

6

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13 February 2020

page(s)

997-1006

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Hyperpolarization-activated cation channels: from genes to function

13 February 2020

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Properties of hyperpolarization-activated pacemaker current defined by coassembly of HCN1 and HCN2 subunits and basal modulation by cyclic nucleotide

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Characterization of the hyperpolarization-activated current, I(f), in ventricular myocytes from human failing heart

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Hyperpolarization-Activated Inward Current in Ventricular Myocytes From Normal and Failing Human Hearts

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Effects of rapid atrial pacing on the arrhythmogenic activity of single cardiomyocytes from pulmonary veins: implication in initiation of atrial fibrillation

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Control of heart rate by cAMP sensitivity of HCN channels

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Cardiac pacemaker function of HCN4 channels in mice is confined to embryonic development and requires cyclic AMP.

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HCN4 provides a 'depolarization reserve' and is not required for heart rate acceleration in mice

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Absence epilepsy and sinus dysrhythmia in mice lacking the pacemaker channel HCN2

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The hyperpolarization-activated HCN1 channel is important for motor learning and neuronal integration by cerebellar Purkinje cells

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Deep bradycardia and heart block caused by inducible cardiac-specific knockout of the pacemaker channel gene Hcn4

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Identifiers

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13 February 2020

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