NF-κB accumulation associated with COL1A1 transactivators defects during chronological aging represses type I collagen expression through a -112/-61-bp region of the COL1A1 promoter in human skin fibroblasts (Q84311088)

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scientific article published on 07 June 2012

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English	NF-κB accumulation associated with COL1A1 transactivators defects during chronological aging represses type I collagen expression through a -112/-61-bp region of the COL1A1 promoter in human skin fibroblasts	scientific article published on 07 June 2012

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3 February 2020

NF-κB accumulation associated with COL1A1 transactivators defects during chronological aging represses type I collagen expression through a -112/-61-bp region of the COL1A1 promoter in human skin fibroblasts (English)

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3 February 2020

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3 February 2020

Karim Boumediene

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3 February 2020

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Gallic Beauchef

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Magalie Hervieu

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3 February 2020

Thierry Oddos

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Magali Demoor

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3 February 2020

Philippe Galéra

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7 June 2012

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Journal of Investigative Dermatology

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2360-2367

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3 February 2020

A rapid micropreparation technique for extraction of DNA-binding proteins from limiting numbers of mammalian cells

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The p65 subunit of NF-κB inhibits COL1A1 gene transcription in human dermal and scleroderma fibroblasts through its recruitment on promoter by protein interaction with transcriptional activators (c-Krox, Sp1, and Sp3).

1 reference

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Age-related changes in the proteoglycans of human skin. Specific cleavage of decorin to yield a major catabolic fragment in adult skin

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Modulation of human alpha1(I) procollagen gene activity by interaction with Sp1 and Sp3 transcription factors in vitro

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Modulation of skin collagen metabolism in aged and photoaged human skin in vivo.

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Age-dependent increases in interstitial collagenase and MAP Kinase levels are exacerbated by superoxide dismutase deficiencies

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High doses of TGF-β potently suppress type I collagen via the transcription factorCUX1

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Aging-induced up-regulation of nuclear binding activities of oxidative stress responsive NF-kB transcription factor in mouse cardiac muscle

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Constitutively phosphorylated Smad3 interacts with Sp1 and p300 in scleroderma fibroblasts

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Phosphorylated extracellular signal-regulated protein kinases 1 and 2 phosphorylate Sp1 on serine 59 and regulate cellular senescence via transcription of p21Sdi1/Cip1/Waf1.

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Human collagen Krox up-regulates type I collagen expression in normal and scleroderma fibroblasts through interaction with Sp1 and Sp3 transcription factors

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Age-dependent remodeling of connective tissue: role of fibronectin and laminin

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Senescence-associated beta-galactosidase is lysosomal beta-galactosidase

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Activation of fibroblast procollagen alpha 1(I) transcription by mechanical strain is transforming growth factor-beta-dependent and involves increased binding of CCAAT-binding factor (CBF/NF-Y) at the proximal promoter

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Transcriptional regulation of cellular ageing by the CCAAT box-binding factor CBF/NF-Y.

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Nuclear factor Y (NF-Y) and cellular senescence

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Aging alters functionally human dermal papillary fibroblasts but not reticular fibroblasts: a new view of skin morphogenesis and aging

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Molecular aspects of skin ageing.

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Downregulation of transcription factor, Sp1, during cellular senescence

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Intrinsic aging- and photoaging-dependent level changes of glycosaminoglycans and their correlation with water content in human skin

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Matrix-degrading metalloproteinases in photoaging

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NF-kappaB inhibits expression of the alpha1(I) collagen gene.

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Activation of nuclear factor-kappa B signalling promotes cellular senescence

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CCAAT binding transcription factor binds and regulates human COL1A1 promoter activity in human dermal fibroblasts: demonstration of increased binding in systemic sclerosis fibroblasts

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NF-kappaB signaling in the aging process

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Genetics vs. entropy: longevity factors suppress the NF-kappaB-driven entropic aging process

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Protecting skin photoaging by NF-kappaB inhibitor

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TGF-beta and TNF-alpha: antagonistic cytokines controlling type I collagen gene expression

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