Myeloproliferative neoplasms cause glomerulopathy (Q84306888)

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3 February 2020

title

Myeloproliferative neoplasms cause glomerulopathy (English)

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2

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author name string

Samar M Said

1

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3 February 2020

Sanjeev Sethi

3

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3 February 2020

Lynn D Cornell

4

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3 February 2020

Mary E Fidler

5

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3 February 2020

Joseph P Grande

6

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3 February 2020

Sandra Herrmann

7

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3 February 2020

Ayalew Tefferi

8

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3 February 2020

Vivette D D'Agati

9

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3 February 2020

Samih H Nasr

10

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language of work or name

English

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publication date

8 June 2011

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3 February 2020

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volume

80

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issue

7

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page(s)

753-759

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Essential thrombocytosis with acute renal failure due to bilateral thrombosis of the renal arteries and veins

3 February 2020

cites work

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Receptor-specific increase in extracellular matrix production in mouse mesangial cells by advanced glycosylation end products is mediated via platelet-derived growth factor

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Glomerular cell proliferation and PDGF expression precede glomerulosclerosis in the remnant kidney model

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Aldose reductase regulates TGF-beta1-induced production of fibronectin and type IV collagen in cultured rat mesangial cells

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Elevated expression of transforming growth factor-beta and proteoglycan production in experimental glomerulonephritis. Possible role in expansion of the mesangial extracellular matrix

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Apoptosis in podocytes induced by TGF-beta and Smad7

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The cyclin-dependent kinase inhibitor p21 is required for TGF-beta1-induced podocyte apoptosis

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Kidney disease associated with plasma cell dyscrasias

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Fibrillary glomerulonephritis and immunotactoid (microtubular) glomerulopathy are associated with distinct immunologic features

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Proposals and rationale for revision of the World Health Organization diagnostic criteria for polycythemia vera, essential thrombocythemia, and primary myelofibrosis: recommendations from an ad hoc international expert panel

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