Passive induction of experimental allergic encephalomyelitis (Q80285957)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:17487182 AND SRC:MED&resulttype=core&format=json

30 December 2019

title

Passive induction of experimental allergic encephalomyelitis (English)

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30 December 2019

2

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30 December 2019

author name string

Ingunn M Stromnes

series ordinal

1

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30 December 2019

publication date

1 January 2006

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30 December 2019

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30 December 2019

volume

1

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30 December 2019

issue

4

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30 December 2019

page(s)

1952-1960

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Immunology of multiple sclerosis

30 December 2019

cites work

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Active induction of experimental allergic encephalomyelitis

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Autoimmunity to myelin oligodendrocyte glycoprotein in rats mimics the spectrum of multiple sclerosis pathology

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Antibody association with a novel model for primary progressive multiple sclerosis: induction of relapsing-remitting and progressive forms of EAE in H2s mouse strains

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A neuropathological analysis of experimental autoimmune encephalomyelitis with predominant brain stem and cerebellar involvement and differences between active and passive induction

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A pathogenic role for myelin-specific CD8( ) T cells in a model for multiple sclerosis

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Transgenic mice that express a myelin basic protein-specific T cell receptor develop spontaneous autoimmunity

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High incidence of spontaneous autoimmune encephalomyelitis in immunodeficient anti-myelin basic protein T cell receptor transgenic mice

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Low avidity recognition of self-antigen by T cells permits escape from central tolerance.

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Transfer of allergic encephalomyelitis in rats by means of lymph node cells

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The rapid isolation of clonable antigen-specific T lymphocyte lines capable of mediating autoimmune encephalomyelitis

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T-cell clones specific for myelin basic protein induce chronic relapsing paralysis and demyelination.

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Migratory Activity and Functional Changes of Green Fluorescent Effector Cells before and during Experimental Autoimmune Encephalomyelitis

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Inflammatory leukocytes and cytokines in the peptide-induced disease of experimental allergic encephalomyelitis in SJL and B10.PL mice

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IL-23 drives a pathogenic T cell population that induces autoimmune inflammation

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Gender, age, and season at immunization uniquely influence the genetic control of susceptibility to histopathological lesions and clinical signs of experimental allergic encephalomyelitis: implications for the genetics of multiple sclerosis

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Treatment of experimental encephalomyelitis with a novel chimeric fusion protein of myelin basic protein and proteolipid protein

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Immune tolerance to myelin proteins.

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