Disparate effects of telomere attrition on gene expression during replicative senescence of human mammary epithelial cells cultured under different conditions (Q80196205)

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scientific article published on 01 August 2004

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English	Disparate effects of telomere attrition on gene expression during replicative senescence of human mammary epithelial cells cultured under different conditions	scientific article published on 01 August 2004

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scholarly article

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Europe PubMed Central

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:15195144 AND SRC:MED&resulttype=core&format=json

29 December 2019

Disparate effects of telomere attrition on gene expression during replicative senescence of human mammary epithelial cells cultured under different conditions (English)

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Europe PubMed Central

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29 December 2019

Brittney-Shea Herbert

2

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Brittney-Shea Herbert

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29 December 2019

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Jerry W Shay

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29 December 2019

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Hong Zhang

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29 December 2019

Kuang-Hung Pan

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29 December 2019

Stanley N Cohen

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29 December 2019

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1 August 2004

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29 December 2019

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29 December 2019

23

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29 December 2019

37

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29 December 2019

6193-6198

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29 December 2019

Switching and signaling at the telomere

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Oxidative DNA damage and senescence of human diploid fibroblast cells.

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Human keratinocytes that express hTERT and also bypass a p16(INK4a)-enforced mechanism that limits life span become immortal yet retain normal growth and differentiation characteristics

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Cluster analysis and display of genome-wide expression patterns

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The telomere terminal transferase of Tetrahymena is a ribonucleoprotein enzyme with two kinds of primer specificity

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Telomere loss: mitotic clock or genetic time bomb?

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The serial cultivation of human diploid cell strains

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p16(INK4a) inactivation is not required to immortalize human mammary epithelial cells.

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Refining the telomere-telomerase hypothesis of aging and cancer

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Immortalization of human CD8 T cell clones by ectopic expression of telomerase reverse transcriptase

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Senescence Induced by Altered Telomere State, Not Telomere Loss

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Specific Association of Human Telomerase Activity with Immortal Cells and Cancer

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Both Rb/p16INK4a inactivation and telomerase activity are required to immortalize human epithelial cells.

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Premature senescence involving p53 and p16 is activated in response to constitutive MEK/MAPK mitogenic signaling

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Telomerase maintains telomere structure in normal human cells.

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Human fibroblast commitment to a senescence-like state in response to histone deacetylase inhibitors is cell cycle dependent

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Analysis of DNA microarrays using algorithms that employ rule-based expert knowledge

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Bypass of telomere-dependent replicative senescence (M1) upon overexpression of Cdk4 in normal human epithelial cells.

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Putative telomere-independent mechanisms of replicative aging reflect inadequate growth conditions

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Agents that cause DNA double strand breaks lead to p16INK4a enrichment and the premature senescence of normal fibroblasts

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Normal human mammary epithelial cells spontaneously escape senescence and acquire genomic changes

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Transfer of the human telomerase reverse transcriptase (TERT) gene into T lymphocytes results in extension of replicative potential

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Senescence as a mode of tumor suppression

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Oncogenic ras provokes premature cell senescence associated with accumulation of p53 and p16INK4a

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Cellular senescence: mitotic clock or culture shock?

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Erosion of the telomeric single-strand overhang at replicative senescence

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Mild Hyperoxia Shortens Telomeres and Inhibits Proliferation of Fibroblasts: A Model for Senescence?

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