Analysis of base excision DNA repair of the oxidative lesion 2-deoxyribonolactone and the formation of DNA-protein cross-links (Q79773158)

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25 December 2019

title

Analysis of base excision DNA repair of the oxidative lesion 2-deoxyribonolactone and the formation of DNA-protein cross-links (English)

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25 December 2019

author name string

Jung-Suk Sung

series ordinal

1

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25 December 2019

Bruce Demple

series ordinal

2

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25 December 2019

publication date

1 January 2006

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25 December 2019

published in

Methods in Enzymology

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25 December 2019

volume

408

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25 December 2019

page(s)

48-64

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Impairment of proliferating cell nuclear antigen-dependent apurinic/apyrimidinic site repair on linear DNA.

25 December 2019

cites work

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Fate of the C-1′ peroxyl radical in the 2′-deoxyuridine system

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Covalent trapping of human DNA polymerase beta by the oxidative DNA lesion 2-deoxyribonolactone

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Dynamics and diversions in base excision DNA repair of oxidized abasic lesions

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The enzymology of apurinic/apyrimidinic endonucleases

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Different DNA polymerases are involved in the short- and long-patch base excision repair in mammalian cells

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The 2-deoxyribonolactone lesion produced in DNA by neocarzinostatin and other damaging agents forms cross-links with the base-excision repair enzyme endonuclease III.

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Flap endonuclease 1 efficiently cleaves base excision repair and DNA replication intermediates assembled into nucleosomes

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The reactivity of the 2-deoxyribonolactone lesion in single-stranded DNA and its implication in reaction mechanisms of DNA damage and repair

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Neocarzinostatin acts as a sensitive probe of DNA microheterogeneity: switching of chemistry from C-1' to C-4' by a G.T mismatch 5' to the site of DNA damage

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Second pathway for completion of human DNA base excision-repair: reconstitution with purified proteins and requirement for DNase IV (FEN1).

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The 7-nitroindole nucleoside as a photochemical precursor of 2'-deoxyribonolactone: access to DNA fragments containing this oxidative abasic lesion

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Site-Specific Generation of Deoxyribonolactone Lesions in DNA Oligonucleotides

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Dynamics of the interaction of human apurinic endonuclease (Ape1) with its substrate and product

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Excision of deoxyribose phosphate residues by DNA polymerase beta during DNA repair

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AP lyases and dRPases: commonality of mechanism

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Mechanism of DNA cleavage by cationic manganese porphyrins: hydroxylations at the 1'-carbon and 5'-carbon atoms of deoxyriboses as initial damages

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Requirement of mammalian DNA polymerase-beta in base-excision repair.

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Mammalian abasic site base excision repair. Identification of the reaction sequence and rate-determining steps

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Fidelity of uracil-initiated base excision DNA repair in Escherichia coli cell extracts

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Long-patch base excision DNA repair of 2-deoxyribonolactone prevents the formation of DNA-protein cross-links with DNA polymerase beta

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7 January 2021

Escherichia coli Uracil- and Ethenocytosine-Initiated Base Excision DNA Repair: Rate-Limiting Step and Patch Size Distribution

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DNA damage induced via independent generation of the radical resulting from formal hydrogen atom abstraction from the C1′-position of a nucleotide

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7 January 2021

Incision Activity of Human Apurinic Endonuclease (Ape) at Abasic Site Analogs in DNA

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7 January 2021

Action of human apurinic endonuclease (Ape1) on C1'-oxidized deoxyribose damage in DNA.

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7 January 2021

Half-life and DNA strand scission products of 2-deoxyribonolactone oxidative DNA damage lesions

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7 January 2021

10.1016/S0076-6879(06)08004-9

Identifiers

DOI

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25 December 2019

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