Astroglia-mediated effects of uric acid to protect spinal cord neurons from glutamate toxicity (Q79489612)

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23 December 2019

title

Astroglia-mediated effects of uric acid to protect spinal cord neurons from glutamate toxicity (English)

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23 December 2019

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23 December 2019

author name string

Yangzhou Du

1

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23 December 2019

Christopher P Chen

2

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23 December 2019

Chia-Yi Tseng

3

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23 December 2019

Yuval Eisenberg

4

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23 December 2019

publication date

1 April 2007

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23 December 2019

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volume

55

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23 December 2019

issue

5

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23 December 2019

page(s)

463-472

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Towards the physiological function of uric acid

23 December 2019

cites work

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7 January 2021

Motor neurons are selectively vulnerable to AMPA/kainate receptor-mediated injury in vitro

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Neurotoxicity of glutamate at the concentration released upon spinal cord injury

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7 January 2021

NMDA and non-NMDA receptor antagonists protect against excitotoxic injury in the rat spinal cord

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Astrocytes block axonal regeneration in mammals by activating the physiological stop pathway

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Changes in amino acid concentrations over time and space around an impact injury and their diffusion through the rat spinal cord

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Glutamate uptake disguises neurotoxic potency of glutamate agonists in cerebral cortex in dissociated cell culture

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Inhibition of neurite outgrowth on astroglial scars in vitro

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The Total Antioxidant Capacity of Human Serum Measured Using Enhanced Chemiluminescence is Almost Completely Accounted for by Urate

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Uric acid protects against secondary damage after spinal cord injury

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7 January 2021

Group I and II metabotropic glutamate receptor expression in cultured rat spinal cord astrocytes

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Regeneration beyond the glial scar

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Reaction of uric acid with peroxynitrite and implications for the mechanism of neuroprotection by uric acid

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SIN-1-induced cytotoxicity in mixed cortical cell culture: peroxynitrite-dependent and -independent induction of excitotoxic cell death.

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Rapid changes in expression of glutamate transporters after spinal cord injury

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Delayed Antagonism of AMPA/Kainate Receptors Reduces Long-Term Functional Deficits Resulting from Spinal Cord Trauma

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Differential regulation of neurotrophin expression in basal forebrain astrocytes by neuronal signals

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Administration of glutamate into the spinal cord at extracellular concentrations reached post-injury causes functional impairments

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7 January 2021

Proteins of the intermediate filament cytoskeleton as markers for astrocytes and human astrocytomas

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7 January 2021

Spinal cord neurons are vulnerable to rapidly triggered kainate neurotoxicity in vitro

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7 January 2021

Uric acid protects neurons against excitotoxic and metabolic insults in cell culture, and against focal ischemic brain injury in vivo

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7 January 2021

Identifiers

DOI

10.1002/GLIA.20472

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:17203476 AND SRC:MED&resulttype=core&format=json

23 December 2019

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