DNA polymerase zeta introduces multiple mutations when bypassing spontaneous DNA damage in Saccharomyces cerevisiae (Q73406613)

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3 November 2019

title

DNA polymerase zeta introduces multiple mutations when bypassing spontaneous DNA damage in Saccharomyces cerevisiae (English)

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3 November 2019

main subject

Saccharomyces cerevisiae

object named as

Saccharomyces cerevisiae

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B D Harfe

1

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3 November 2019

S Jinks-Robertson

series ordinal

2

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3 November 2019

publication date

1 December 2000

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3 November 2019

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3 November 2019

volume

6

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3 November 2019

issue

6

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3 November 2019

page(s)

1491-1499

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Specific cleavage of model recombination and repair intermediates by the yeast Rad1-Rad10 DNA endonuclease

3 November 2019

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Physical interaction between components of DNA mismatch repair and nucleotide excision repair

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Mismatch repair proteins regulate heteroduplex formation during mitotic recombination in yeast

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Defective transcription-coupled repair of oxidative base damage in Cockayne syndrome patients from XP group G

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The function of the human homolog of Saccharomyces cerevisiae REV1 is required for mutagenesis induced by UV light

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Yeast Rad7-Rad16 Complex, Specific for the Nucleotide Excision Repair of the Nontranscribed DNA Strand, Is an ATP-dependent DNA Damage Sensor

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Yeast DNA-repair gene RAD14 encodes a zinc metalloprotein with affinity for ultraviolet-damaged DNA.

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Substrate spectrum of human excinuclease: repair of abasic sites, methylated bases, mismatches, and bulky adducts

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RAD1 and RAD10, but not other excision repair genes, are required for double-strand break-induced recombination in Saccharomyces cerevisiae

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Eukaryotic polymerases iota and zeta act sequentially to bypass DNA lesions

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Repair of DNA loops involves DNA-mismatch and nucleotide-excision repair proteins

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Base excision repair of oxidative DNA damage activated by XPG protein

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DNA repair protects against cutaneous and internal neoplasia: evidence from xeroderma pigmentosum

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Identifiers

DOI

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3 November 2019

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