Effects of deregulated Raf activation on integrin, cytokine-receptor expression and the induction of apoptosis in hematopoietic cells (Q73166100)

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1 November 2019

title

Effects of deregulated Raf activation on integrin, cytokine-receptor expression and the induction of apoptosis in hematopoietic cells (English)

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1 November 2019

4

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1 November 2019

Carla Burrows

5

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1 November 2019

author name string

C Weinstein-Oppenheimer

1

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1 November 2019

L S Steelman

2

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1 November 2019

P A Algate

3

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1 November 2019

P E Hoyle

6

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1 November 2019

J T Lee

7

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1 November 2019

P W Moye

8

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1 November 2019

J G Shelton

9

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1 November 2019

R Franklin

10

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1 November 2019

J A McCubrey

11

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1 November 2019

publication date

1 November 2000

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1 November 2019

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1 November 2019

volume

14

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1 November 2019

issue

11

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1 November 2019

page(s)

1921-1938

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Signal transduction, cell cycle regulatory, and anti-apoptotic pathways regulated by IL-3 in hematopoietic cells: possible sites for intervention with anti-neoplastic drugs

1 November 2019

cites work

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Cytokine receptors and signal transduction

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Growth of factor-dependent hemopoietic precursor cell lines

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Enrichment of hematopoietic precursor cells and cloning of multipotential B-lymphocyte precursors

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Establishment and characterization of a unique human cell line that proliferates dependently on GM-CSF, IL-3, or erythropoietin

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Differential abilities of activated Raf oncoproteins to abrogate cytokine dependency, prevent apoptosis and induce autocrine growth factor synthesis in human hematopoietic cells

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A conditionally-active form of MEK1 results in autocrine tranformation of human and mouse hematopoietic cells.

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Effects of deregulated RAF and MEK1 expression on the cytokine-dependency of hematopoietic cells.

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Combined effects of aberrant MEK1 activity and BCL2 overexpression on relieving the cytokine dependency of human and murine hematopoietic cells.

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Signaling through the hematopoietic cytokine receptors.

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The roles of signaling by the p42/p44 mitogen-activated protein (MAP) kinase pathway; a potential route to radio- and chemo-sensitization of tumor cells resulting in the induction of apoptosis and loss of clonogenicity

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Post-natal lethality and neurological and gastrointestinal defects in mice with targeted disruption of the A-Raf protein kinase gene.

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Raf revealed in life-or-death decisions

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Endothelial apoptosis in Braf-deficient mice

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Craf-1 protein kinase is essential for mouse development

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Conditionally oncogenic forms of the A-Raf and B-Raf protein kinases display different biological and biochemical properties in NIH 3T3 cells

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Mutations of critical amino acids affect the biological and biochemical properties of oncogenic A-Raf and Raf-1

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Differential abilities of the Raf family of protein kinases to abrogate cytokine dependency and prevent apoptosis in murine hematopoietic cells by a MEK1-dependent mechanism

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Synergy between Raf and BCL2 in abrogating the cytokine dependency of hematopoietic cells.

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Serine/threonine phosphorylation in cytokine signal transduction

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The effect of activating mutations on dimerization, tyrosine phosphorylation and internalization of the macrophage colony stimulating factor receptor

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A phosphatidylinositol 3-kinase-dependent pathway that differentially regulates c-Raf and A-Raf

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The human beta 2 integrin CD18 promoter consists of two inverted Ets cis elements

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Identification of cell-specific and developmentally regulated nuclear factors that direct myeloid and lymphoid expression of the CD11a gene

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The promoter of the CD11b gene directs myeloid-specific and developmentally regulated expression

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Characterization of subsets of bone marrow-derived macrophages by flow cytometry analysis

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