Thapsigargin-evoked changes in human platelet Ca2 , Na , pH and membrane potential (Q72578498)

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27 October 2019

title

Thapsigargin-evoked changes in human platelet Ca2 , Na , pH and membrane potential (English)

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:8229793 AND SRC:MED&resulttype=core&format=json

27 October 2019

author name string

M Kimura

series ordinal

1

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:8229793 AND SRC:MED&resulttype=core&format=json

27 October 2019

N Lasker

series ordinal

2

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:8229793 AND SRC:MED&resulttype=core&format=json

27 October 2019

A Aviv

series ordinal

3

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:8229793 AND SRC:MED&resulttype=core&format=json

27 October 2019

language of work or name

English

0 references

publication date

1 May 1993

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:8229793 AND SRC:MED&resulttype=core&format=json

27 October 2019

number of pages

13

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inferred from page(s)

published in

Journal of Physiology

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27 October 2019

volume

464

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27 October 2019

page(s)

1-13

1 reference

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Intracellular pH measurements in Ehrlich ascites tumor cells utilizing spectroscopic probes generated in situ

27 October 2019

cites work

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The inflammatory and tumor-promoting sesquiterpene lactone, thapsigargin, activates platelets by selective mobilization of calcium as shown by protein phosphorylations

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The calcium mobilizing tumor promoting agent, thapsigargin elevates the platelet cytoplasmic free calcium concentration to a higher steady state level. A possible mechanism of action for the tumor promotion

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Activation of calcium entry by the tumor promoter thapsigargin in parotid acinar cells. Evidence that an intracellular calcium pool and not an inositol phosphate regulates calcium fluxes at the plasma membrane

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Thapsigargin, a novel molecular probe for studying intracellular calcium release and storage

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Thapsigargin, a tumor promoter, discharges intracellular Ca2 stores by specific inhibition of the endoplasmic reticulum Ca2( )-ATPase

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Voltage-gated potassium channels and the control of membrane potential in human platelets

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Ca2 influx in human T lymphocytes is induced independently of inositol phosphate production by mobilization of intracellular Ca2 stores. A study with the Ca2 endoplasmic reticulum-ATPase inhibitor thapsigargin

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Agonist-evoked alkaline shift in the cytosolic pH set point for activation of Na /H antiport in human platelets. The role of cytosolic Ca2 and protein kinase C.

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The calcium paradox in isolated guinea-pig ventricular myocytes: effects of membrane potential and intracellular sodium

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Further characterization of the mechanisms mediating the rise in cytosolic free Na in thrombin-stimulated platelets. Evidence for inhibition of the Na ,K( )-ATPase and for Na entry via a Ca2 influx pathway

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Inhibition of the sarcoplasmic reticulum Ca2 transport ATPase by thapsigargin at subnanomolar concentrations

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Cytosolic and stored calcium antagonistically control tyrosine phosphorylation of specific platelet proteins

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Thapsigargin inhibits the sarcoplasmic or endoplasmic reticulum Ca-ATPase family of calcium pumps

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Different calcium pools in human platelets and their role in thromboxane A2 formation

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Drug action of thapsigargin on the Ca2 pump protein of sarcoplasmic reticulum

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Agonist-induced Ca2 influx into human platelets is secondary to the emptying of intracellular Ca2 stores

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Endothelin-induced calcium responses in human vascular smooth muscle cells

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Cyclic nucleotides attenuate thrombin-evoked alterations in parameters of platelet Na/H antiport. The role of cytosolic Ca

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Mechanism of ion permeation through calcium channels

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