Nerve sprouting capacity in a pharmacologically induced mouse model of spinal muscular atrophy. (Q64997231)

3 July 2019

title

Nerve sprouting capacity in a pharmacologically induced mouse model of spinal muscular atrophy. (English)

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3 July 2019

main subject

spinal muscular atrophy

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6

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3 July 2019

author name string

Mendell Rimer

1

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3 July 2019

Bonnie L Seaberg

2

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3 July 2019

Pei-Fen Yen

3

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3 July 2019

Steven Lam

4

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3 July 2019

Robert Louis Hastings

5

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3 July 2019

Wesley J Thompson

7

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3 July 2019

Zhihua Feng

8

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3 July 2019

Friedrich Metzger

9

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3 July 2019

Sergey Paushkin

10

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3 July 2019

Chien-Ping Ko

11

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3 July 2019

language of work or name

English

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publication date

24 May 2019

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3 July 2019

published in

Scientific Reports

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3 July 2019

volume

9

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3 July 2019

issue

1

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3 July 2019

page(s)

7799

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3 July 2019

Incidence, prevalence, and gene frequency studies of chronic childhood spinal muscular atrophy

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A single nucleotide in the SMN gene regulates splicing and is responsible for spinal muscular atrophy

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SMNDelta7, the major product of the centromeric survival motor neuron (SMN2) gene, extends survival in mice with spinal muscular atrophy and associates with full-length SMN

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Pharmacologically induced mouse model of adult spinal muscular atrophy to evaluate effectiveness of therapeutics after disease onset

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Motor neuron disease. SMN2 splicing modifiers improve motor function and longevity in mice with spinal muscular atrophy

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Neuromuscular synaptogenesis: coordinating partners with multiple functions

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Perisynaptic Schwann Cells at the Neuromuscular Synapse: Adaptable, Multitasking Glial Cells

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Terminal Schwann cells participate in neuromuscular synapse remodeling during reinnervation following nerve injury

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Terminal Schwann cells elaborate extensive processes following denervation of the motor endplate

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Nerve sprouting in muscle is induced and guided by processes extended by Schwann cells

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Motor nerve sprouting

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Ciliary neurotrophic factor-induced sprouting preserves motor function in a mouse model of mild spinal muscular atrophy

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Compensatory axon sprouting for very slow axonal die-back in a transgenic model of spinal muscular atrophy type III.

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Neurofilament accumulation at the motor endplate and lack of axonal sprouting in a spinal muscular atrophy mouse model

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Defects in neuromuscular junction remodelling in the Smn(2B/-) mouse model of spinal muscular atrophy

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Reduced survival motor neuron (Smn) gene dose in mice leads to motor neuron degeneration: an animal model for spinal muscular atrophy type III

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Smn, the spinal muscular atrophy-determining gene product, modulates axon growth and localization of beta-actin mRNA in growth cones of motoneurons

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Synaptic Defects in the Spinal and Neuromuscular Circuitry in a Mouse Model of Spinal Muscular Atrophy

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Identifiers

DOI

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3 July 2019

PMC publication ID

6534600

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3 July 2019

PubMed publication ID

31127156

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3 July 2019