Periostin Mediates Right Ventricular Failure through Induction of Inducible Nitric Oxide Synthase Expression in Right Ventricular Fibroblasts from Monocrotaline-Induced Pulmonary Arterial Hypertensive Rats (Q60934909)

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journal article from 'International Journal of Molecular Sciences' published in 2019

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English	Periostin Mediates Right Ventricular Failure through Induction of Inducible Nitric Oxide Synthase Expression in Right Ventricular Fibroblasts from Monocrotaline-Induced Pulmonary Arterial Hypertensive Rats	journal article from 'International Journal of Molecular Sciences' published in 2019

Statements

scholarly article

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Periostin Mediates Right Ventricular Failure through Induction of Inducible Nitric Oxide Synthase Expression in Right Ventricular Fibroblasts from Monocrotaline-Induced Pulmonary Arterial Hypertensive Rats (English)

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author name string

Keisuke Imoto

1

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Muneyoshi Okada

2

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Hideyuki Yamawaki

3

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language of work or name

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publication date

24 December 2018

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January 2019

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full work available at URL

https://www.mdpi.com/1422-0067/20/1/62

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International Journal of Molecular Sciences

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20

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1

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62

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copyright license

Creative Commons Attribution 4.0 International

24 December 2018

1 reference

April 2022 Public Data File from Crossref

copyright status

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Genetic manipulation of periostin expression reveals a role in cardiac hypertrophy and ventricular remodeling

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Periostin is essential for cardiac healing after acute myocardial infarction

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Periostin promotes a fibroblastic lineage pathway in atrioventricular valve progenitor cells

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Nitric oxide and nitric oxide synthase isoforms in the normal, hypertrophic, and failing heart

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Periostin is a novel factor in cardiac remodeling after experimental and clinical unloading of the failing heart.

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Periostin advances atherosclerotic and rheumatic cardiac valve degeneration by inducing angiogenesis and MMP production in humans and rodents.

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Survival in patients with idiopathic, familial, and anorexigen-associated pulmonary arterial hypertension in the modern management era.

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Periostin promotes chronic allergic inflammation in response to Th2 cytokines.

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Stimulating myocardial regeneration with periostin Peptide in large mammals improves function post-myocardial infarction but increases myocardial fibrosis

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Right ventricular diastolic impairment in patients with pulmonary arterial hypertension

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Periostin expression is upregulated and associated with myocardial fibrosis in human failing hearts.

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Pressure-overload-induced right heart failure

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10.3390/IJMS20010062

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