Suppression of Apoptosis in Human Umbilical Vein Endothelial Cells (HUVECs) by Klotho Protein is Associated with Reduced Endoplasmic Reticulum Oxidative Stress and Activation of the PI3K/AKT Pathway (Q60312905)

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article published in 2018

Language	Label	Description	Also known as
English	Suppression of Apoptosis in Human Umbilical Vein Endothelial Cells (HUVECs) by Klotho Protein is Associated with Reduced Endoplasmic Reticulum Oxidative Stress and Activation of the PI3K/AKT Pathway	article published in 2018

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scholarly article

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Suppression of Apoptosis in Human Umbilical Vein Endothelial Cells (HUVECs) by Klotho Protein is Associated with Reduced Endoplasmic Reticulum Oxidative Stress and Activation of the PI3K/AKT Pathway (English)

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endoplasmic reticulum

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based on heuristic

inferred from title

object named as

Bin Leng

2

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author name string

Wei Cui

1

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Wei Liu

3

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GaoPin Wang

4

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language of work or name

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publication date

24 November 2018

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number of pages

11

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inferred from page(s)

Medical Science Monitor

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24

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8489-8499

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CHOP is implicated in programmed cell death in response to impaired function of the endoplasmic reticulum

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Interactions between the PI3K and Raf signaling pathways can result in the transformation of hematopoietic cells

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Endothelial cell apoptosis: biochemical characteristics and potential implications for atherosclerosis

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Coupling endoplasmic reticulum stress to the cell death program: role of the ER chaperone GRP78

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Critical role of endogenous Akt/IAPs and MEK1/ERK pathways in counteracting endoplasmic reticulum stress-induced cell death

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Peroxynitrite causes endoplasmic reticulum stress and apoptosis in human vascular endothelium: implications in atherogenesis

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Anti-apoptotic and anti-senescence effects of Klotho on vascular endothelial cells

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Constant and intermittent high glucose enhances endothelial cell apoptosis through mitochondrial superoxide overproduction

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ER stress in cardiovascular disease

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Attenuation of NF-kappaB and activation of Nrf2 signaling by 1,2,4-triazine derivatives, protects neuron-like PC12 cells against apoptosis

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Endoplasmic reticulum stress as a therapeutic target in cardiovascular disease

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The Role of Endoplasmic Reticulum Stress and Unfolded Protein Response in Atherosclerosis

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Epidemiology of Atherosclerosis and the Potential to Reduce the Global Burden of Atherothrombotic Disease.

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PI3K-Akt-mTOR signal inhibition affects expression of genes related to endoplasmic reticulum stress

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Multivesicular body formation enhancement and exosome release during endoplasmic reticulum stress.

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Identifiers

10.12659/MSM.911202

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