Metabolic reprogramming of natural killer cells in obesity limits antitumor responses (Q59319272)

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3 May 2020

title

Metabolic reprogramming of natural killer cells in obesity limits antitumor responses (English)

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Andrew Hogan

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Semir Beyaz

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Cathriona Foley

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Cliona O'Farrelly

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Barbara S Nikolajczyk

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Kingston H Mills

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Michael B Brenner

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David K. Finlay

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Lydia Lynch

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author name string

Xavier Michelet

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Danielle Duquette

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Kevin Wei

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Ali Tavakkoli

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Raymond Donnelly

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Mathilde Raverdeau

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Ashley Vernon

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William Pettee

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Roisin M Loftus

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Donal O'Shea

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language of work or name

English

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publication date

12 November 2018

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volume

19

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issue

12

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3 May 2020

page(s)

1330-1340

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Overweight, obesity and cancer: epidemiological evidence and proposed mechanisms

3 May 2020

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mTORC1-dependent metabolic reprogramming is a prerequisite for NK cell effector function.

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The metabolic checkpoint kinase mTOR is essential for IL-15 signaling during the development and activation of NK cells

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Maternal obesity drives functional alterations in uterine NK cells.

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PDK1 regulation of mTOR and hypoxia-inducible factor 1 integrate metabolism and migration of CD8 T cells

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Two modes of lytic granule fusion during degranulation by natural killer cells

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Secretory cytotoxic granule maturation and exocytosis require the effector protein hMunc13-4

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Identifiers

DOI

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3 May 2020

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