Protein kinase D1 deletion in adipocytes enhances energy dissipation and protects against adiposity (Q58616122)

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title

Protein kinase D1 deletion in adipocytes enhances energy dissipation and protects against adiposity (English)

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Alexander Mayer

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Jonathan Trujillo Viera

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Miguel Marcos

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Guadalupe Sabio

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Martin Eilers

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Grzegorz Sumara

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author name string

Angel Loza Valdes

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Rabih El-Merahbi

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Carsten P Ade

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Till Karwen

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Werner Schmitz

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Anja Slotta

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Manuela Erk

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Sudha Janaki-Raman

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Nuria Matesanz

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Jorge L Torres

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Almut Schulze

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language of work or name

English

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publication date

2 November 2018

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volume

37

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issue

22

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ImageJ

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11 June 2022

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inferred from PubMed Central ID database lookup

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PKA phosphorylates and inactivates AMPKalpha to promote efficient lipolysis

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Protein kinase D1 and D2 are involved in chemokine release induced by toll-like receptors 2, 4, and 5.

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G protein-coupled receptor (GPR)40-dependent potentiation of insulin secretion in mouse islets is mediated by protein kinase D1.

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Protein kinase-D1 overexpression prevents lipid-induced cardiac insulin resistance

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PKD1 Inhibits AMPKα2 through Phosphorylation of Serine 491 and Impairs Insulin Signaling in Skeletal Muscle Cells

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Deletion of Protein Kinase D1 in Pancreatic Beta Cells Impairs Insulin Secretion in High-Fat Fed Mice

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Identifiers

DOI

10.15252/EMBJ.201899182

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:30389661 AND SRC:MED&resulttype=core&format=json

1 May 2020

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1 May 2020

PubMed publication ID

30389661

1 reference