Apc deficiency predisposes to renal carcinoma in the mouse (Q57247904)

5

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Owen J Sansom

object named as

Owen J Sansom

1

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Douglas J Winton

4

object named as

Douglas J Winton

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author name string

David F R Griffiths

Beta-catenin and TCF mediate cell positioning in the intestinal epithelium by controlling the expression of EphB/ephrinB

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language of work or name

English

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publication date

22 August 2005

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published in

Oncogene

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volume

24

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issue

55

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page(s)

8205-8210

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cites work

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Somatic mutations of the APC gene in primary breast cancers

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Inactivation of Apc perturbs mammary development, but only directly results in acanthoma in the context of Tcf-1 deficiency.

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Inducible Cre-mediated control of gene expression in the murine gastrointestinal tract: effect of loss of beta-catenin

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Identification of FAP locus genes from chromosome 5q21

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Susceptibility to vascular neoplasms but no increased susceptibility to renal carcinogenesis in Vhl knockout mice.

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A germ-line Tsc1 mutation causes tumor development and embryonic lethality that are similar, but not identical to, those caused by Tsc2 mutation in mice

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Nuclear accumulation of beta-catenin protein in Wilms' tumours

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Tsc2( /-) mice develop tumors in multiple sites that express gelsolin and are influenced by genetic background

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Loss of heterozygosity and protein expression of APC gene in renal cell carcinomas.

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Cystic renal neoplasia following conditional inactivation of apc in mouse renal tubular epithelium

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Loss of Apc in vivo immediately perturbs Wnt signaling, differentiation, and migration

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Rapid colorectal adenoma formation initiated by conditional targeting of the Apc gene.

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Generalized lacZ expression with the ROSA26 Cre reporter strain

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Aberrant expression of beta-catenin and mutation of exon 3 of the beta-catenin gene in renal and urothelial carcinomas.

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Anaplastic Wilms' tumour, a subtype displaying poor prognosis, harbours p53 gene mutations

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inferred from DOI database lookup

7 January 2021

based on heuristic

Early development of polycystic kidney disease in transgenic mice expressing an activated mutant of the beta-catenin gene

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inferred from DOI database lookup

7 January 2021

based on heuristic

Overexpression of human Dickkopf-1, an antagonist of wingless/WNT signaling, in human hepatoblastomas and Wilms' tumors.

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