Genetic ablation of Rac1 in cartilage results in chondrodysplasia. (Q53556500)

15 May 2018

title

Genetic ablation of Rac1 in cartilage results in chondrodysplasia. (English)

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15 May 2018

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Frank Beier

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15 May 2018

author name string

Guoyan Wang

1

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15 May 2018

Anita Woods

2

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15 May 2018

Hanga Agoston

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15 May 2018

Veronica Ulici

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15 May 2018

language of work or name

English

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publication date

1 April 2007

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15 May 2018

published in

Developmental Biology

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15 May 2018

volume

306

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15 May 2018

issue

2

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15 May 2018

page(s)

612-623

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15 May 2018

Regulation of Rho family GTPases by cell-cell and cell-matrix adhesion.

cites work

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Regulation of osteoclast apoptosis and motility by small GTPase binding protein Rac1.

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Rac1 deletion in mouse neutrophils has selective effects on neutrophil functions.

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Rac1 modulates sphingosine 1-phosphate-mediated activation of phosphoinositide 3-kinase/Akt signaling pathways in vascular endothelial cells

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Integrin-linked kinase regulates chondrocyte shape and proliferation

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Microarray analyses of gene expression during chondrocyte differentiation identifies novel regulators of hypertrophy

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External GTP-bound transglutaminase 2 is a molecular switch for chondrocyte hypertrophic differentiation and calcification.

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The collagen receptor DDR2 regulates proliferation and its elimination leads to dwarfism

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RhoA/ROCK signaling suppresses hypertrophic chondrocyte differentiation.

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7 January 2021

Identifiers

DOI

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15 May 2018

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15 May 2018