Gluthatione level is altered in lymphoblasts from patients with familial Alzheimer's disease. (Q53231519)

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2 November 2019

title

Gluthatione level is altered in lymphoblasts from patients with familial Alzheimer's disease (English)

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2 November 2019

1

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2 November 2019

Sandro Sorbi

3

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2 November 2019

author name string

S Latorraca

2

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2 November 2019

T Iantomasi

4

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2 November 2019

F Favilli

5

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2 November 2019

M T Vincenzini

6

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2 November 2019

G Liguri

7

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2 November 2019

language of work or name

English

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publication date

1 November 1999

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2 November 2019

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2 November 2019

volume

275

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2 November 2019

issue

2

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2 November 2019

page(s)

152-154

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Neurodegenerative disorders in humans: the role of glutathione in oxidative stress-mediated neuronal death

2 November 2019

cites work

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High-performance liquid chromatography of thiols and disulfides: dinitrophenol derivatives

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Mitochondrial function, GSH and iron in neurodegeneration and Lewy body diseases.

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Alzheimer's presenilin mutation sensitizes neural cells to apoptosis induced by trophic factor withdrawal and amyloid beta-peptide: involvement of calcium and oxyradicals.

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Amyloid, the presenilins and Alzheimer's disease

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Glutathione metabolism in Crohn's disease

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Increased Peroxidation and Reduced Antioxidant Enzyme Activity in Alzheimer's Disease

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Amyloid beta-peptide impairs ion-motive ATPase activities: evidence for a role in loss of neuronal Ca2 homeostasis and cell death

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Presenilins, the Endoplasmic Reticulum, and Neuronal Apoptosis in Alzheimer's Disease

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Amino acids, glutathione, and glutathione transferase activity in the brains of patients with Alzheimer's disease.

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Cellular mechanisms of resistance to chronic oxidative stress

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Cell biology of the amyloid beta-protein precursor and the mechanism of Alzheimer's disease

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Involvement of free radicals in dementia of the Alzheimer type: a hypothesis

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Participation of presenilin 2 in apoptosis: enhanced basal activity conferred by an Alzheimer mutation

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7 January 2021

10.1016/S0304-3940(99)00751-X

Identifiers

DOI

1 reference

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2 November 2019

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