Selective PDE5A inhibition with sildenafil rescues left ventricular dysfunction, inflammatory immune response and cardiac remodeling in angiotensin II-induced heart failure in vivo. (Q52886208)

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scientific article published on 2 November 2012

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English	Selective PDE5A inhibition with sildenafil rescues left ventricular dysfunction, inflammatory immune response and cardiac remodeling in angiotensin II-induced heart failure in vivo.	scientific article published on 2 November 2012

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16 February 2020

Selective PDE5A inhibition with sildenafil rescues left ventricular dysfunction, inflammatory immune response and cardiac remodeling in angiotensin II-induced heart failure in vivo (English)

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16 February 2020

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based on heuristic

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ventricular dysfunction

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based on heuristic

inferred from title

left ventricular dysfunction

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based on heuristic

inferred from title

3

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16 February 2020

Matthias Fröhlich

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16 February 2020

Carsten Tschöpe

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Carsten Tschöpe

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16 February 2020

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Dirk Westermann

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16 February 2020

Peter Moritz Becher

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16 February 2020

Kostantinos Savvatis

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16 February 2020

Yu Xia

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16 February 2020

Sebastian Hoffmann

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16 February 2020

Heinz-Peter Schultheiss

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16 February 2020

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2 November 2012

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16 February 2020

Basic Research in Cardiology

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16 February 2020

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16 February 2020

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16 February 2020

308

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16 February 2020

Validation of oligonucleotide microarray data using microfluidic low-density arrays: a new statistical method to normalize real-time RT-PCR data

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Role of heart rate reduction in the prevention of experimental heart failure: comparison between If-channel blockade and β-receptor blockade.

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Prognostic significance of left ventricular mass change during treatment of hypertension

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Chemokines and cardiac fibrosis.

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Phosphodiesterase-5 inhibition with sildenafil attenuates cardiomyocyte apoptosis and left ventricular dysfunction in a chronic model of doxorubicin cardiotoxicity

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Sildenafil and phosphodiesterase-5 inhibitors for heart failure

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Long-term use of sildenafil in the therapeutic management of heart failure

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Sildenafil citrate increases myocardial cGMP content in rat heart, decreases its hypertrophic response to isoproterenol and decreases myocardial leak of creatine kinase and troponin T

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21 January 2018

Inflammation as a therapeutic target in heart failure? A scientific statement from the Translational Research Committee of the Heart Failure Association of the European Society of Cardiology

1 reference

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21 January 2018

Effect of sildenafil on cardiac performance in patients with heart failure

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Chronic treatment with long acting phosphodiesterase-5 inhibitor tadalafil alters proteomic changes associated with cytoskeletal rearrangement and redox regulation in Type 2 diabetic hearts.

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ADAMTS-1 is an active metalloproteinase associated with the extracellular matrix

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Potential novel pharmacological therapies for myocardial remodelling

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Potential therapeutic targets for cardiac fibrosis: TGFbeta, angiotensin, endothelin, CCN2, and PDGF, partners in fibroblast activation

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PDE5A suppression of acute beta-adrenergic activation requires modulation of myocyte beta-3 signaling coupled to PKG-mediated troponin I phosphorylation

1 reference

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Purification of an insect derived recombinant human ADAMTS-1 reveals novel gelatin (type I collagen) degrading activities.

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Small-molecule therapies for cardiac hypertrophy: moving beneath the cell surface

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Cyclic nucleotide phosphodiesterase 1A: a key regulator of cardiac fibroblast activation and extracellular matrix remodeling in the heart

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Neonatal calyceal dilation and renal fibrosis resulting from loss of Adamts-1 in mouse kidney is due to a developmental dysgenesis

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21 January 2018

Tackling heart failure in the twenty-first century

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21 January 2018

Sildenafil stops progressive chamber, cellular, and molecular remodeling and improves calcium handling and function in hearts with pre-existing advanced hypertrophy caused by pressure overload

1 reference

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21 January 2018

Phosphodiesterase type 5 is highly expressed in the hypertrophied human right ventricle, and acute inhibition of phosphodiesterase type 5 improves contractility

1 reference

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Toll-like receptor-4 deficiency attenuates doxorubicin-induced cardiomyopathy in mice

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Central role of RAGE-dependent neointimal expansion in arterial restenosis

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Sildenafil (Viagra) attenuates ischemic cardiomyopathy and improves left ventricular function in mice

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Cardiac Uses of Phosphodiesterase-5 Inhibitors

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21 January 2018

Chronic inhibition of cyclic GMP phosphodiesterase 5A prevents and reverses cardiac hypertrophy

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21 January 2018

Novel therapeutic targets for the treatment of heart failure.

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Antiproliferative effect of sildenafil on human pulmonary artery smooth muscle cells

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S100A8/A9 aggravates post-ischemic heart failure through activation of RAGE-dependent NF-κB signaling

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