Differential Effector Engagement by Oncogenic KRAS. (Q49997178)

PubMed publication ID

29444439

23 February 2018

http://europepmc.org/abstract/MED/29444439

title

Differential Effector Engagement by Oncogenic KRAS (English)

1 reference

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13 May 2020

4

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3 June 2020

Christof Fellmann

25

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3 June 2020

Andrew M Waters

11

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15

Patricia Greninger

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8

Leslie A Strathern

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14

Rosy Liao

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16

Giovanna T Stein

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17

Ellen Murchie

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18

Eliane Cortez

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author name string

Tina L Yuan

1

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3 June 2020

Arnaud Amzallag

2

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Rachel Bagni

3

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Shervin Afghani

5

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William Burgan

6

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Nicole Fer

7

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Katie Powell

9

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Brian Smith

10

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Ty Thomson

13

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Regina K Egan

19

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Lauren Procter

20

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Matthew Bess

21

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Kwong Tai Cheng

22

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Chih-Shia Lee

23

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Liam Changwoo Lee

24

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Robert Stephens

26

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Ji Luo

27

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3 June 2020

Scott W Lowe

28

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3 June 2020

Cyril H Benes

29

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Frank McCormick

30

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David Drubin

12

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publication date

1 February 2018

1 reference

PubMed publication ID

29444439

23 February 2018

http://europepmc.org/abstract/MED/29444439

published in

Cell Reports

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volume

22

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issue

7

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3 June 2020

page(s)

1889-1902

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describes a project that uses

3 June 2020

limma

1 reference

22 June 2022

https://europepmc.org/article/PMC/6343826

inferred from PubMed Central ID database lookup

cites work

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Ras-dependent carbon metabolism and transformation in mouse fibroblasts

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Requirement for Rac1 in a K-ras induced lung cancer in the mouse.

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mTOR controls mitochondrial oxidative function through a YY1-PGC-1alpha transcriptional complex

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A gene expression signature associated with "K-Ras addiction" reveals regulators of EMT and tumor cell survival

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Requirement for NF-kappaB signalling in a mouse model of lung adenocarcinoma

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Systematic RNA interference reveals that oncogenic KRAS-driven cancers require TBK1

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mTOR regulation of autophagy

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Autophagy facilitates glycolysis during Ras-mediated oncogenic transformation

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Activated Ras requires autophagy to maintain oxidative metabolism and tumorigenesis.

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Functional identification of optimized RNAi triggers using a massively parallel sensor assay

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Oncogenic and wild-type Ras play divergent roles in the regulation of mitogen-activated protein kinase signaling

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mTORC1 controls mitochondrial activity and biogenesis through 4E-BP-dependent translational regulation.

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Oncogene ablation-resistant pancreatic cancer cells depend on mitochondrial function

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Enhanced MET Translation and Signaling Sustains K-Ras-Driven Proliferation under Anchorage-Independent Growth Conditions

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Co-occurring genomic alterations define major subsets of KRAS-mutant lung adenocarcinoma with distinct biology, immune profiles, and therapeutic vulnerabilities

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AMPK Inhibits the Stimulatory Effects of TGF-β on Smad2/3 Activity, Cell Migration, and Epithelial-to-Mesenchymal Transition

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Selective Inhibition of Oncogenic KRAS Output with Small Molecules Targeting the Inactive State

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The energy sensor AMPK regulates Hedgehog signaling in human cells through a unique Gli1 metabolic checkpoint

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Epithelial-to-Mesenchymal Transition Defines Feedback Activation of Receptor Tyrosine Kinase Signaling Induced by MEK Inhibition in KRAS-Mutant Lung Cancer

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10.1016/J.CELREP.2018.01.051

Identifiers

DOI

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:29444439 AND SRC:MED&resulttype=core&format=json

release_d3saohtl6fho5hcwp33hu5zdki

3 June 2020

Fatcat ID

1 reference

Fatcat

https://api.fatcat.wiki/v0/release/d3saohtl6fho5hcwp33hu5zdki

24 November 2022

mapped directly with Wikidata item

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3 June 2020

PubMed publication ID

29444439

1 reference