p53 and Bax activation in 6-hydroxydopamine-induced apoptosis in PC12 cells (Q48765497)

15 February 2018

title

p53 and Bax activation in 6-hydroxydopamine-induced apoptosis in PC12 cells (English)

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15 February 2018

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1

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author name string

Y Wu

2

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M F Nissou

3

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S Arnaud

4

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Alim-Louis-Benabid

5

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J M Verna

6

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language of work or name

English

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publication date

1 March 1997

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15 February 2018

published in

Brain Research

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15 February 2018

volume

751

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issue

1

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page(s)

139-142

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The Bcl-2 family of proteins, and the regulation of neuronal survival

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Cellular responses to DNA damage: cell-cycle checkpoints, apoptosis and the roles of p53 and ATM

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bcl-x is expressed in embryonic and postnatal neural tissues and functions to prevent neuronal cell death

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Complex I inhibitors induce dose-dependent apoptosis in PC12 cells: relevance to Parkinson's disease

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7 January 2021

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Upregulation of bax protein levels in neurons following cerebral ischemia.

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The protooncogene bcl-2 inhibits apoptosis in PC12 cells

7 January 2021

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Tumor suppressor p53 is a direct transcriptional activator of the human bax gene

7 January 2021

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Rapid colorimetric assay for cellular growth and survival: Application to proliferation and cytotoxicity assays

7 January 2021

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Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programmed cell death

7 January 2021

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p53 induction is associated with neuronal damage in the central nervous system

7 January 2021

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Nuclear accumulation of p53 protein following kainic acid-induced seizures.

7 January 2021

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Inflammatory mediator stimulation of astrocytes and meningeal fibroblasts induces neuronal degeneration via the nitridergic pathway

7 January 2021

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Neurotoxin-induced cell death in neuronal PC12 cells is mediated by induction of apoptosis

7 January 2021

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The role of free radicals and p53 in neuron apoptosis in vivo

7 January 2021

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Bad, a heterodimeric partner for Bcl-XL and Bcl-2, displaces Bax and promotes cell death

7 January 2021

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10.1016/S0006-8993(96)01358-3

7 January 2021

Identifiers

DOI

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15 February 2018

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15 February 2018