Requirement for mitogen-activated protein kinase in cerebellar long term depression (Q48222676)

9 February 2018

title

Requirement for mitogen-activated protein kinase in cerebellar long term depression (English)

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9 February 2018

author

Hiroshi Kawasaki

1

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Hiroaki Fujii

2

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author name string

Y Gotoh

3

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T Morooka

4

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S Shimohama

5

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E Nishida

6

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T Hirano

7

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language of work or name

English

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publication date

1 May 1999

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Journal of Biological Chemistry

published in

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volume

274

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issue

19

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page(s)

13498-13502

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How MAP kinases are regulated

cites work

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The MAP kinase cascade is essential for diverse signal transduction pathways

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ERKs: a family of protein-serine/threonine kinases that are activated and tyrosine phosphorylated in response to insulin and NGF

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Calcium is an intracellular mediator of the climbing fiber in induction of cerebellar long-term depression

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Participation of Postsynaptic PKC in Cerebellar Long-Term Depression in Culture

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Stimulation of Protein Tyrosine Phosphorylation by NMDA Receptor Activation

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MAP kinase translocates into the nucleus of the presynaptic cell and is required for long-term facilitation in Aplysia.

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Mutation in the phosphorylation sites of MAP kinase blocks learning-related internalization of apCAM in Aplysia sensory neurons.

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A requirement for the mitogen-activated protein kinase cascade in hippocampal long term potentiation.

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Antibodies inactivating mGluR1 metabotropic glutamate receptor block long-term depression in cultured Purkinje cells

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Membrane depolarization and calcium influx stimulate MEK and MAP kinase via activation of Ras.

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Ras-dependent and Ras-independent activation pathways for the stress-activated-protein-kinase cascade

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Differential pre- and postsynaptic mechanisms for synapic potentiation and depression between a granule cell and a purkinje cell in rat cerebellar culture

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PD 098059 is a specific inhibitor of the activation of mitogen-activated protein kinase kinase in vitro and in vivo

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Deficient cerebellar long-term depression and impaired motor learning in mGluR1 mutant mice

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Purification and identification of a major activator for p38 from osmotically shocked cells. Activation of mitogen-activated protein kinase kinase 6 by osmotic shock, tumor necrosis factor-alpha, and H2O2

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Ca(2 )-dependent routes to Ras: mechanisms for neuronal survival, differentiation, and plasticity?

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Characterization of multiple phosphorylation sites on the AMPA receptor GluR1 subunit

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A protein synthesis-dependent late phase of cerebellar long-term depression

21 January 2018

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Activation and involvement of p38 mitogen-activated protein kinase in glutamate-induced apoptosis in rat cerebellar granule cells

21 January 2018

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21 January 2018

Identifiers

DOI

10.1074/JBC.274.19.13498

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9 February 2018

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9 February 2018