CD95 ligand (Fas-L/APO-1L) and tumor necrosis factor-related apoptosis-inducing ligand mediate ischemia-induced apoptosis in neurons (Q48218375)

9 February 2018

title

CD95 ligand (Fas-L/APO-1L) and tumor necrosis factor-related apoptosis-inducing ligand mediate ischemia-induced apoptosis in neurons (English)

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9 February 2018

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Klaus-Michael Debatin

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author name string

Martin-Villalba A

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Herr I

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Jeremias I

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Hahne M

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Brandt R

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Vogel J

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Schenkel J

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Herdegen T

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language of work or name

English

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publication date

1 May 1999

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9 February 2018

published in

Journal of Neuroscience

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volume

19

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issue

10

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page(s)

3809-3817

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9 February 2018

Identification of programmed cell death in situ via specific labeling of nuclear DNA fragmentation

cites work

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12 December 2020

JNK, but not MAPK, activation is associated with Fas-mediated apoptosis in human T cells

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Comparison between the timing of JNK activation, c-Jun phosphorylation, and onset of death commitment in sympathetic neurones.

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Procedural and strain-related variables significantly affect outcome in a murine model of focal cerebral ischemia

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c-Jun can recruit JNK to phosphorylate dimerization partners via specific docking interactions

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Tumor necrosis factor-alpha gene transfer augments anti-Fas antibody-mediated apoptosis in human glioma cells

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Stimulation of c-Jun kinase and mitogen-activated protein kinase by ischemia and reperfusion in the perfused rat heart

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Lasting N-terminal phosphorylation of c-Jun and activation of c-Jun N-terminal kinases after neuronal injury.

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FK506 and the role of immunophilins in nerve regeneration

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DNA damaging agents induce expression of Fas ligand and subsequent apoptosis in T lymphocytes via the activation of NF-kappa B and AP-1.

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Mitogen-activated protein kinase-mediated Fas apoptotic signaling pathway

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Effect of brain edema on infarct volume in a focal cerebral ischemia model in rats

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Early delineation of ischemic tissue in rat brain cryosections by high-contrast staining

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Localization of Fas antigen mRNA induced in postischemic murine forebrain by in situ hybridization

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Reversible middle cerebral artery occlusion without craniectomy in rats

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Induction of apoptosis by the transcription factor c-Jun

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Activation of CD95 (APO-1/Fas) signaling by ceramide mediates cancer therapy-induced apoptosis

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Mitochondrial control of apoptosis

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Immunophilins and nervous system

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Immunophilins mediate the neuroprotective effects of FK506 in focal cerebral ischaemia

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A semiautomated method for measuring brain infarct volume.

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The c-Jun N-terminal kinase cascade plays a role in stress-induced apoptosis in Jurkat cells by up-regulating Fas ligand expression.

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Colon carcinoma cells use different mechanisms to escape CD95-mediated apoptosis

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Fas expression on human fetal astrocytes without susceptibility to fas-mediated cytotoxicity

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Identification of an oncoprotein- and UV-responsive protein kinase that binds and potentiates the c-Jun activation domain

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Regulation of the human TNF promoter by the transcription factor Ets.

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Evidence supporting a role for programmed cell death in focal cerebral ischemia in rats.

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Global ischemia can cause DNA fragmentation indicative of apoptosis in rat brain

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Opposite functions of jun-B and c-jun in growth regulation and neuronal differentiation

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Suppression of signalling through transcription factor NF-AT by interactions between calcineurin and Bcl-2.

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The stress-activated protein kinase subfamily of c-Jun kinases

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TRAIL/Apo-2-ligand-induced apoptosis in human T cells.

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The immunosuppressant FK506 ameliorates ischaemic damage in the rat brain

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Induction of DNA fragmentation after 10 to 120 minutes of focal cerebral ischemia in rats

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Persistent activation of c-Jun N-terminal kinase 1 (JNK1) in gamma radiation-induced apoptosis

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A c-Jun dominant negative mutant protects sympathetic neurons against programmed cell death

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The jun proto-oncogene is positively autoregulated by its product, Jun/AP-1

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The Immunosuppressant Drug FK506 Ameliorates Secondary Mitochondrial Dysfunction Following Transient Focal Cerebral Ischemia in the Rat

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Role of the Jun Kinase Pathway in the Regulation of c-Jun Expression and Apoptosis in Sympathetic Neurons

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Apoptosis by death factor

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Phosphorylation of c-Jun Is Necessary for Apoptosis Induced by Survival Signal Withdrawal in Cerebellar Granule Neurons

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Poly(ADP-ribose) polymerase gene disruption renders mice resistant to cerebral ischemia

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Monitoring of CD95 (APO-1/Fas) ligand expression in human T cells by quantitative RT-PCR

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Activation of c-Jun N-terminal kinase during ischemia and reperfusion in mouse liver

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Transient cerebral ischemia. Association of apoptosis induction with hypoperfusion

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Activation-induced cell death in murine T cell hybridomas. Differential regulation of Fas (CD95) versus Fas ligand expression by cyclosporin A and FK506.

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Absence of excitotoxicity-induced apoptosis in the hippocampus of mice lacking the Jnk3 gene

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12 December 2020

Fas and Fas ligand in embryos and adult mice: ligand expression in several immune-privileged tissues and coexpression in adult tissues characterized by apoptotic cell turnover

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12 December 2020

Pure, postmitotic, polarized human neurons derived from NTera 2 cells provide a system for expressing exogenous proteins in terminally differentiated neurons

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12 December 2020

Nitric oxide mediates glutamate neurotoxicity in primary cortical cultures

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12 December 2020

Early increases in TNF-alpha, IL-6 and IL-1 beta levels following transient cerebral ischemia in gerbil brain

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12 December 2020

Mouse strain differences in susceptibility to cerebral ischemia are related to cerebral vascular anatomy

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12 December 2020

Identifiers

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9 February 2018