Transport of BMAA into Neurons and Astrocytes by System xc. (Q48194642)

9 February 2018

http://europepmc.org/abstract/MED/28470569

title

Transport of BMAA into Neurons and Astrocytes by System xc. (English)

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9 February 2018

http://europepmc.org/abstract/MED/28470569

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2

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:28470569 AND SRC:MED&resulttype=core&format=json

9 June 2020

author name string

Rebecca Albano

series ordinal

1

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:28470569 AND SRC:MED&resulttype=core&format=json

9 June 2020

publication date

3 May 2017

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9 February 2018

http://europepmc.org/abstract/MED/28470569

published in

Neurotoxicity Research

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:28470569 AND SRC:MED&resulttype=core&format=json

9 June 2020

volume

33

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:28470569 AND SRC:MED&resulttype=core&format=json

9 June 2020

issue

1

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9 June 2020

page(s)

1-5

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Oxidative stress, glutamate, and neurodegenerative disorders

9 June 2020

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The natural non-protein amino acid N-β-methylamino-L-alanine (BMAA) is incorporated into protein during synthesis

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Selective brain uptake and behavioral effects of the cyanobacterial toxin BMAA (beta-N-methylamino-L-alanine) following neonatal administration to rodents

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Selective death of cholinergic neurons induced by beta-methylamino-L-alanine

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Beta-N-methylamino-L-alanine enhances neurotoxicity through multiple mechanisms

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Time-dependent changes in striatal xCT protein expression in hemi-Parkinson rats

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Dopaminergic neurons of system x(c)⁻-deficient mice are highly protected against 6-hydroxydopamine-induced toxicity.

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System xC- is a mediator of microglial function and its deletion slows symptoms in amyotrophic lateral sclerosis mice

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Insulin-like growth factor 1 and transforming growth factor-β stimulate cystine/glutamate exchange activity in dental pulp cells

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System Xc- and apolipoprotein E expressed by microglia have opposite effects on the neurotoxicity of amyloid-beta peptide 1-40.

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BMAA selectively injures motor neurons via AMPA/kainate receptor activation

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Specific antagonism of excitotoxic action of 'uncommon' amino acids assayed in organotypic mouse cortical cultures

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Facilitated transport of the neurotoxin, beta-N-methylamino-L-alanine, across the blood-brain barrier

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21 January 2018

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Cortical neurones with Ca2 permeable AMPA/kainate channels display distinct receptor immunoreactivity and are GABAergic

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Antioxidants protect against glutamate-induced cytotoxicity in a neuronal cell line

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12 December 2020

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System x(c)- activity and astrocytes are necessary for interleukin-1 beta-mediated hypoxic neuronal injury

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Macromolecular synthesis inhibitors prevent oxidative stress-induced apoptosis in embryonic cortical neurons by shunting cysteine from protein synthesis to glutathione.

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Glutamate toxicity in a neuronal cell line involves inhibition of cystine transport leading to oxidative stress

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Immature cortical neurons are uniquely sensitive to glutamate toxicity by inhibition of cystine uptake

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12 December 2020

In vitro kainate injury to large, SMI-32( ) spinal neurons is Ca2 dependent.

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12 December 2020