Changes in biochemical processes in cerebellar granule cells of mice exposed to methylmercury (Q48138134)

9 February 2018

title

Changes in biochemical processes in cerebellar granule cells of mice exposed to methylmercury (English)

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4

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author name string

Sairam Bellum

1

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Bhupinder Bawa

2

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Kerry A Thuett

3

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Louise C Abbott

5

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publication date

1 May 2007

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International Journal of Toxicology

published in

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volume

26

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issue

3

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page(s)

261-269

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Methylmercury Poisoning in Iraq

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Coordination deficits induced in young adult mice treated with methylmercury

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Translocation of apoptosis-inducing factor in cerebellar granule cells exposed to neurotoxic agents inducing oxidative stress

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Fluoro-jade identification of cerebellar granule cell and purkinje cell death in the α1A calcium ion channel mutant mouse, leaner

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Pharmacological characterization of the effects of methylmercury and mercuric chloride on spontaneous noradrenaline release from rat hippocampal slices

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Methylmercury cytotoxicity in PC12 cells is mediated by primary glutathione depletion independent of excess reactive oxygen species generation

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Mercuric compounds inhibit human monocyte function by inducing apoptosis: evidence for formation of reactive oxygen species, development of mitochondrial membrane permeability transition and loss of reductive reserve

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Effect of thimerosal, a preservative in vaccines, on intracellular Ca2 concentration of rat cerebellar neurons

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Serofendic acid, a neuroprotective substance derived from fetal calf serum, inhibits mitochondrial membrane depolarization and caspase-3 activation

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Acute exposure to methylmercury opens the mitochondrial permeability transition pore in rat cerebellar granule cells

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Evidence for interactions between intracellular calcium stores during methylmercury-induced intracellular calcium dysregulation in rat cerebellar granule neurons

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Pathways Mediating CaEntry in Rat Cerebellar Granule Cells FollowingExposure to Methyl Mercury

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Elevations of Intracellular Ca2 as a Probable Contributor to Decreased Viability in Cerebellar Granule Cells Following Acute Exposure to Methylmercury

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Methylmercury poisoning of the developing nervous system: morphological changes in neuronal mitochondria

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Cytochrome c release precedes mitochondrial membrane potential loss in cerebellar granule neuron apoptosis: lack of mitochondrial swelling

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Biochemical changes in the brain in rats poisoned with an alkymercury compound, with special reference to the inhibition of protein synthesis in brain cortex slices

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21 January 2018

Identifiers

DOI

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9 February 2018

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9 February 2018