The activation level of the TNF family receptor, Edar, determines cusp number and tooth number during tooth development. (Q47925750)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:15031115 AND SRC:MED&resulttype=core&format=json

25 December 2019

title

The activation level of the TNF family receptor, Edar, determines cusp number and tooth number during tooth development (English)

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25 December 2019

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1

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25 December 2019

Paul A Overbeek

4

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25 December 2019

Jo-Maree Courtney

3

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25 December 2019

Paul T Sharpe

5

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25 December 2019

author name string

Headon DJ

2

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25 December 2019

language of work or name

English

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publication date

1 April 2004

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25 December 2019

published in

Developmental Biology

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25 December 2019

volume

268

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25 December 2019

issue

1

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25 December 2019

page(s)

185-194

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X-linked anhidrotic ectodermal dysplasia with immunodeficiency is caused by impaired NF-kappaB signaling

25 December 2019

cites work

1 reference

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Ectodysplasin is released by proteolytic shedding and binds to the EDAR protein

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Cloning of Tabby, the murine homolog of the human EDA gene: evidence for a membrane-associated protein with a short collagenous domain.

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Involvement of a novel Tnf receptor homologue in hair follicle induction

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Gene defect in ectodermal dysplasia implicates a death domain adapter in development

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Ectodysplasin receptor-mediated signaling is essential for embryonic submandibular salivary gland development

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X-linked anhidrotic (hypohidrotic) ectodermal dysplasia is caused by mutation in a novel transmembrane protein

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Signaling and subcellular localization of the TNF receptor Edar

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Postnatal lower jaw dentition in different phenotypes of tabby mice

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The ectodermal dysplasia receptor activates the nuclear factor-kappaB, JNK, and cell death pathways and binds to ectodysplasin A

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TNF signaling via the ligand-receptor pair ectodysplasin and edar controls the function of epithelial signaling centers and is regulated by Wnt and activin during tooth organogenesis

7 January 2021

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Identification of a new splice form of the EDA1 gene permits detection of nearly all X-linked hypohidrotic ectodermal dysplasia mutations

7 January 2021

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Mutations in the human homologue of mouse dl cause autosomal recessive and dominant hypohidrotic ectodermal dysplasia

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Stimulation of ectodermal organ development by Ectodysplasin-A1.

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Traf6 is essential for murine tooth cusp morphogenesis

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Cusp patterning defect in Tabby mouse teeth and its partial rescue by FGF.

7 January 2021

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The teeth of the “sleek” mouse

7 January 2021

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The Tabby phenotype is caused by mutation in a mouse homologue of the EDA gene that reveals novel mouse and human exons and encodes a protein (ectodysplasin-A) with collagenous domains

7 January 2021

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Ectodysplasin-A1 is sufficient to rescue both hair growth and sweat glands in Tabby mice

7 January 2021

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Expression of cytokeratin 14 in ameloblast-lineage cells of the developing tooth of rat, both in vivo and in vitro.

7 January 2021

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Tissue-specific and differentiation-specific expression of a human K14 keratin gene in transgenic mice

7 January 2021

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