Cold Environment Exacerbates Brain Pathology and Oxidative Stress Following Traumatic Brain Injuries: Potential Therapeutic Effects of Nanowired Antioxidant Compound H-290/51. (Q47258212)

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scientific article published on 30 August 2017

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English	Cold Environment Exacerbates Brain Pathology and Oxidative Stress Following Traumatic Brain Injuries: Potential Therapeutic Effects of Nanowired Antioxidant Compound H-290/51.	scientific article published on 30 August 2017

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28 March 2020

Cold Environment Exacerbates Brain Pathology and Oxidative Stress Following Traumatic Brain Injuries: Potential Therapeutic Effects of Nanowired Antioxidant Compound H-290/51 (English)

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28 March 2020

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Jose-vicente Lafuente

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28 March 2020

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Aruna Sharma

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28 March 2020

Dafin F Muresanu

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Per-Ove Sjöquist

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28 March 2020

Ranjana Patnaik

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28 March 2020

Z Ryan Tian

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Asya Ozkizilcik

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Hari S Sharma

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1 January 2018

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Molecular Neurobiology

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28 March 2020

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28 March 2020

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28 March 2020

Pathophysiology of Blood-Brain Barrier in Brain Injury in Cold and Hot Environments: Novel Drug Targets for Neuroprotection

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Military-related traumatic brain injury and neurodegeneration

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Multiple traumatic brain injury and concussive symptoms among deployed military personnel

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21 January 2018

Mild hyperthermia worsens the neuropathological damage associated with mild traumatic brain injury in rats

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21 January 2018

Hyperthermia and central nervous system injury

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Permeability of the blood-brain barrier depends on brain temperature

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Blood-brain barrier pathophysiology in traumatic brain injury

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Blood-brain barrier dysfunction following traumatic brain injury

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21 January 2018

Oxidative stress in traumatic brain injury

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Interactions of oxidative stress and neurovascular inflammation in the pathogenesis of traumatic brain injury

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21 January 2018

Cold defence responses: the role of oxidative stress

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Energy budget, oxidative stress and antioxidant in striped hamster acclimated to moderate cold and warm temperatures

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Exacerbation of Methamphetamine Neurotoxicity in Cold and Hot Environments: Neuroprotective Effects of an Antioxidant Compound H-290/51.

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21 January 2018

Nanoparticles Exacerbate Both Ubiquitin and Heat Shock Protein Expressions in Spinal Cord Injury: Neuroprotective Effects of the Proteasome Inhibitor Carfilzomib and the Antioxidant Compound H-290/51.

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21 January 2018

Chronic treatment with nanoparticles exacerbate hyperthermia induced blood-brain barrier breakdown, cognitive dysfunction and brain pathology in the rat. Neuroprotective effects of nanowired-antioxidant compound H-290/51.

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21 January 2018

Role of histamine in traumatic brain edema. An experimental study in the rat.

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21 January 2018

Involvement of nitric oxide in the pathophysiology of acute heat stress in the rat. Influence of a new antioxidant compound H-290/51.

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21 January 2018

A new antioxidant compound H-290/51 modulates glutamate and GABA immunoreactivity in the rat spinal cord following trauma

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21 January 2018

Nanowired drug delivery to enhance neuroprotection in spinal cord injury

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Nanowired Drug Delivery Across the Blood-Brain Barrier in Central Nervous System Injury and Repair.

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Measuring myeloperoxidase activity in biological samples

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Influence of long-term acute heat exposure on regional blood-brain barrier permeability, cerebral blood flow and 5-HT level in conscious normotensive young rats

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Effect of captopril (a converting enzyme inhibitor) on blood-brain barrier permeability and cerebral blood flow in normotensive rats

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Methods to produce brain hyperthermia

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Methods to produce hyperthermia-induced brain dysfunction

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The blood-brain barrier to proteins under normal and pathological conditions

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The relationship between edema, blood-brain-barrier and tissue elements in a local brain injury

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Role of extracellular proteins in the dynamics of vasogenic brain edema

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Presidental address. Neuropathological aspects of brain edema

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Albumin activates astrocytes and microglia through mitogen-activated protein kinase pathways

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Albumin activates the canonical TGF receptor–smad signaling pathway but this is not required for activation of astrocytes

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Selective increase in TNF alpha permeation across the blood-spinal cord barrier after SCI.

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Drugs of abuse-induced hyperthermia, blood-brain barrier dysfunction and neurotoxicity: neuroprotective effects of a new antioxidant compound H-290/51.

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Spinal cord injury induced heat shock protein expression is reduced by an antioxidant compound H-290/51. An experimental study using light and electron microscopy in the rat.

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A new antioxidant compound H-290/51 attenuates nitric oxide synthase and heme oxygenase expression following hyperthermic brain injury. An experimental study using immunohistochemistry in the rat.

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Ambient temperature and development of traumatic brain oedema in anaesthetized animals

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Measurement of vascular reactive oxygen species production by chemiluminescence.

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Characterization of novel indenoindoles. Part I. Structure-activity relationships in different model systems of lipid peroxidation

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12 December 2020

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A new antioxidant compound H-290151 attenuates spinal cord injury induced expression of constitutive and inducible isoforms of nitric oxide synthase and edema formation in the rat.

1 reference

https://pubmed.ncbi.nlm.nih.gov/28856566

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Nitric oxide and carbon monoxide in the brain pathology of heat stress

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Post-injury treatment with a new antioxidant compound H-290/51 attenuates spinal cord trauma-induced c-fos expression, motor dysfunction, edema formation, and cell injury in the rat.

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10.1007/S12035-017-0740-Y

1 reference

Europe PubMed Central

PubMed publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:28856566 AND SRC:MED&resulttype=core&format=json

28 March 2020

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1 reference

Europe PubMed Central

PubMed publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:28856566 AND SRC:MED&resulttype=core&format=json

28 March 2020

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