MyD88 signaling in brain endothelial cells is essential for the neuronal activity and glucocorticoid release during systemic inflammation (Q46818741)

25 December 2017

title

MyD88 signaling in brain endothelial cells is essential for the neuronal activity and glucocorticoid release during systemic inflammation (English)

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25 December 2017

author name string

D Gosselin

series ordinal

1

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25 December 2017

S Rivest

series ordinal

2

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25 December 2017

language of work or name

English

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publication date

8 January 2008

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25 December 2017

published in

Molecular Psychiatry

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25 December 2017

volume

13

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25 December 2017

issue

5

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25 December 2017

page(s)

480-497

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25 December 2017

How circulating cytokines trigger the neural circuits that control the hypothalamic-pituitary-adrenal axis

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Cyclooxygenase 2 (COX-2) inhibition increases the inflammatory response in the brain during systemic immune stimuli

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Endothelial cells of the rat brain vasculature express cyclooxygenase-2 mRNA in response to systemic interleukin-1 beta: a possible site of prostaglandin synthesis responsible for fever

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Targeted disruption of the MyD88 gene results in loss of IL-1- and IL-18-mediated function

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ACTH response induced by interleukin-1 is mediated by CRF secretion stimulated by hypothalamic PGE.

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In altering the release of glucocorticoids, ketorolac exacerbates the effects of systemic immune stimuli on expression of proinflammatory genes in the brain

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Microinjection of prostaglandin E2 and muscimol into the preoptic area in conscious rats: comparison of effects on plasma adrenocorticotrophic hormone (ACTH), body temperature, locomotor activity, and cardiovascular function.

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Ventromedial preoptic prostaglandin E2 activates fever-producing autonomic pathways.

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The febrile response to lipopolysaccharide is blocked in cyclooxygenase-2(-/-), but not in cyclooxygenase-1(-/-) mice

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Cyclooxygenase-2 mediates the febrile response of mice to interleukin-1beta

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Irradiation does not compromise or exacerbate the innate immune response in the brains of mice that were transplanted with bone marrow stem cells

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Identifiers

DOI

10.1038/SJ.MP.4002122

1 reference

25 December 2017

1 reference

25 December 2017