Role of ROS-mediated autophagy in radiation-induced bystander effect of hepatoma cells. (Q46777704)

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29 February 2020

title

Role of ROS-mediated autophagy in radiation-induced bystander effect of hepatoma cells (English)

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29 February 2020

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hepatocellular carcinoma

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7

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29 February 2020

author name string

Xiangdong Wang

1

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29 February 2020

Jianghong Zhang

2

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29 February 2020

Jiamei Fu

3

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29 February 2020

Juan Wang

4

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29 February 2020

Shuang Ye

5

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29 February 2020

Weili Liu

6

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publication date

24 February 2015

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International Journal of Radiation Biology

29 February 2020

published in

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volume

91

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issue

5

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page(s)

452-458

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Cloning and genomic organization of beclin 1, a candidate tumor suppressor gene on chromosome 17q21

29 February 2020

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Up-regulation of ROS by mitochondria-dependent bystander signaling contributes to genotoxicity of bystander effects.

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Radiation and chemotherapy bystander effects induce early genomic instability events: telomere shortening and bridge formation coupled with mitochondrial dysfunction.

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Radiation-induced intercellular signaling mediated by cytochrome-c via a p53-dependent pathway in hepatoma cells

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Heavy ion irradiation induces autophagy in irradiated C2C12 myoblasts and their bystander cells.

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LC3, a mammalian homologue of yeast Apg8p, is localized in autophagosome membranes after processing

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Suppressive effect of ascorbic acid on the mutagenesis induced by the bystander effect through mitochondrial function

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Alpha particle-induced bystander effect is mediated by ROS via a p53-dependent SCO2 pathway in hepatoma cells.

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Production of a signal by irradiated cells which leads to a response in unirradiated cells characteristic of initiation of apoptosis

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Autophagy suppresses tumor progression by limiting chromosomal instability

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Autophagy suppresses tumorigenesis through elimination of p62

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Induction of radioresistance by a nitric oxide-mediated bystander effect

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Signaling factors for irradiated glioma cells induced bystander responses in fibroblasts.

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Bystander signaling between glioma cells and fibroblasts targeted with counted particles.

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Bystander effect induced by counted high-LET particles in confluent human fibroblasts: a mechanistic study.

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21 January 2018

Identifiers

DOI

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:25651038 AND SRC:MED&resulttype=core&format=json

29 February 2020

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