Novel role of the CXC chemokine receptor 3 in inflammatory response to arterial injury: involvement of mTORC1. (Q46212010)

20 December 2017

retracted paper

2 references

Dataset for "Continued use of retracted papers: Temporal trends in citations and (lack of) awareness of retractions shown in citation contexts in biomedicine"

Continued use of retracted papers: Temporal trends in citations and (lack of) awareness of retractions shown in citation contexts in biomedicine

title

Novel role of the CXC chemokine receptor 3 in inflammatory response to arterial injury: involvement of mTORC1. (English)

1 reference

20 December 2017

main subject

inflammation

0 references

author

Hans-Henning Eckstein

6

1 reference

20 December 2017

Nicolas Langwieser

2

object named as

Nicolas Langwieser

1 reference

20 December 2017

8

Albert Schömig

1 reference

20 December 2017

Johannes B K Schwarz

1

1 reference

20 December 2017

Nicole N Langwieser

3

1 reference

20 December 2017

Martin J Bek

4

1 reference

20 December 2017

Stefan Seidl

5

1 reference

20 December 2017

Bao Lu

7

1 reference

20 December 2017

Hermann Pavenstädt

9

1 reference

20 December 2017

Dietlind Zohlnhöfer

10

1 reference

20 December 2017

publication date

4 December 2008

1 reference

20 December 2017

published in

Circulation Research

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20 December 2017

volume

104

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20 December 2017

issue

2

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20 December 2017

page(s)

189-200

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20 December 2017

EMAP-II downregulation contributes to the beneficial effects of rapamycin after vascular injury

cites work

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Differential expression of three T lymphocyte-activating CXC chemokines by human atheroma-associated cells

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Crucial Role of Stromal Cell–Derived Factor-1α in Neointima Formation After Vascular Injury in Apolipoprotein E–Deficient Mice

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A mouse model of vascular injury that induces rapid onset of medial cell apoptosis followed by reproducible neointimal hyperplasia

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Absence of CD40 signaling is associated with an increase in intimal thickening after arterial injury

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Regulation of translation initiation by FRAP/mTOR

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T lymphocytes inhibit the vascular response to injury

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The specific role of chemokines in atherosclerosis.

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Blockade of IP-10/CXCR3 promotes progressive renal fibrosis

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Cell cycle-dependent expression of CXC chemokine receptor 3 by endothelial cells mediates angiostatic activity

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Antibody-mediated blockade of the CXCR3 chemokine receptor results in diminished recruitment of T helper 1 cells into sites of inflammation

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Superoxide production in vascular smooth muscle contributes to oxidative stress and impaired relaxation in atherosclerosis

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Transcriptome analysis reveals a role of interferon-gamma in human neointima formation.

1 reference

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Reduction of atherosclerosis in mice by inhibition of CD40 signalling

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Regulatory effects of mammalian target of rapamycin-activated pathways in type I and II interferon signaling.

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Angiogenesis-Dependent and Independent Phases of Intimal Hyperplasia

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Rapamycin attenuates atherosclerosis induced by dietary cholesterol in apolipoprotein-deficient mice through a p27 Kip1 -independent pathway

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Transplanted human bone marrow contributes to vascular endothelium

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Chemokines take centre stage in vascular biology

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Differential expression of chemokine receptors and chemotactic responsiveness of type 1 T helper cells (Th1s) and Th2s

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TOR signaling in growth and metabolism

1 reference

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Rapamycin effects transcriptional programs in smooth muscle cells controlling proliferative and inflammatory properties

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Up-regulation of early growth response gene-1 via the CXCR3 receptor induces reactive oxygen species and inhibits Na /K -ATPase activity in an immortalized human proximal tubule cell line

1 reference

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Comparison of various bone marrow fractions in the ability to participate in vascular remodeling after mechanical injury

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Sirolimus-Eluting Stents versus Standard Stents in Patients with Stenosis in a Native Coronary Artery

1 reference

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CXCR3 expression on CD34( ) hemopoietic progenitors induced by granulocyte-macrophage colony-stimulating factor: II. Signaling pathways involved

1 reference

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Hematopoietic stem cells differentiate into vascular cells that participate in the pathogenesis of atherosclerosis

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30 December 2023

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Identifiers

DOI

10.1161/CIRCRESAHA.108.182683

1 reference

20 December 2017

1 reference

20 December 2017