Reversible inactivation of superoxide-sensitive aconitase in Abeta1-42-treated neuronal cell lines (Q46117954)

19 December 2017

title

Reversible inactivation of superoxide-sensitive aconitase in Abeta1-42-treated neuronal cell lines (English)

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19 December 2017

author name string

Longo VD

1

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19 December 2017

Viola KL

2

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19 December 2017

Klein WL

3

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19 December 2017

Finch CE

4

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19 December 2017

language of work or name

English

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publication date

1 November 2000

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19 December 2017

Journal of Neurochemistry

published in

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19 December 2017

volume

75

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19 December 2017

issue

5

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19 December 2017

page(s)

1977-1985

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19 December 2017

Both oxidative stress-dependent and independent effects of amyloid beta protein are detected by 3-(4,5-dimethylthiazol-2-yl)-2, 5-diphenyltetrazolium bromide (MTT) reduction assay

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Superoxide radical and superoxide dismutases

21 January 2018

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Aconitase is a sensitive and critical target of oxygen poisoning in cultured mammalian cells and in rat lungs

21 January 2018

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Superoxide radical and iron modulate aconitase activity in mammalian cells

21 January 2018

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Re-examination and further development of a precise and rapid dye method for measuring cell growth/cell kill

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The carboxy terminus of the beta amyloid protein is critical for the seeding of amyloid formation: implications for the pathogenesis of Alzheimer's disease

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Clinical, pathological, and neurochemical changes in dementia: A subgroup with preserved mental status and numerous neocortical plaques

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Superoxide accelerates DNA damage by elevating free-iron levels

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Water-soluble Abeta (N-40, N-42) oligomers in normal and Alzheimer disease brains

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Diffusible, nonfibrillar ligands derived from Abeta1-42 are potent central nervous system neurotoxins

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Cell death induced by β-amyloid 1–40 in MES 23.5 hybrid clone: the role of nitric oxide and NMDA-gated channel activation leading to apoptosis

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Clusterin (apoJ) Alters the Aggregation of Amyloid β-Peptide (Aβ1-42) and Forms Slowly Sedimenting Aβ Complexes That Cause Oxidative Stress

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Requirement for superoxide in excitotoxic cell death

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Alzheimer's disease: genetic studies and transgenic models

21 January 2018

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Morphology and toxicity of Abeta-(1-42) dimer derived from neuritic and vascular amyloid deposits of Alzheimer's disease

21 January 2018

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Translating cell biology into therapeutic advances in Alzheimer's disease

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Iron accumulation in Alzheimer disease is a source of redox-generated free radicals

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Amyloid-beta aggregation: selective inhibition of aggregation in mixtures of amyloid with different chain lengths

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10.1046/J.1471-4159.2000.0751977.X

21 January 2018

Identifiers

DOI

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19 December 2017

1 reference

19 December 2017