Vascular endothelial growth factor (VEGF), produced by feline infectious peritonitis (FIP) virus-infected monocytes and macrophages, induces vascular permeability and effusion in cats with FIP. (Q45367551)

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scientific article published on 5 April 2011

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English	Vascular endothelial growth factor (VEGF), produced by feline infectious peritonitis (FIP) virus-infected monocytes and macrophages, induces vascular permeability and effusion in cats with FIP.	scientific article published on 5 April 2011

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scholarly article

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27 January 2020

Vascular endothelial growth factor (VEGF), produced by feline infectious peritonitis (FIP) virus-infected monocytes and macrophages, induces vascular permeability and effusion in cats with FIP (English)

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27 January 2020

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feline infectious peritonitis

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based on heuristic

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27 January 2020

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Taku Ohyama

2

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27 January 2020

Aiko Kokumoto

3

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27 January 2020

Ryoichi Satoh

4

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27 January 2020

Tsutomu Hohdatsu

5

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27 January 2020

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5 April 2011

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27 January 2020

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27 January 2020

158

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27 January 2020

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27 January 2020

161-168

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27 January 2020

Genetics and pathogenesis of feline infectious peritonitis virus

1 reference

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Feline infectious peritonitis: insights into feline coronavirus pathobiogenesis and epidemiology based on genetic analysis of the viral 3c gene.

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Vascular permeability factor/vascular endothelial growth factor and the significance of microvascular hyperpermeability in angiogenesis

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Vascular permeability factor/endothelial growth factor (VPF/VEGF): accumulation and expression in human synovial fluids and rheumatoid synovial tissue

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IL-6 stimulates Th2 type cytokine secretion and upregulates VEGF and NRP-1 expression in pancreatic cancer cells

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Hantaviruses direct endothelial cell permeability by sensitizing cells to the vascular permeability factor VEGF, while angiopoietin 1 and sphingosine 1-phosphate inhibit hantavirus-directed permeability

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Antibody and cytokine responses in kittens during the development of feline infectious peritonitis (FIP)

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Systemic vascular lesions in feline infectious peritonitis

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A study on the mechanism of antibody-dependent enhancement of feline infectious peritonitis virus infection in feline macrophages by monoclonal antibodies

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Enhancement and Neutralization of Feline Infectious Peritonitis Virus Infection in Feline Macrophages by Neutralizing Monoclonal Antibodies Recognizing Different Epitopes

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Vascular endothelial growth factor immunoneutralization plus Paclitaxel markedly reduces tumor burden and ascites in athymic mouse model of ovarian cancer

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Morphologic features and development of granulomatous vasculitis in feline infectious peritonitis.

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Disease outcome and cytokine responses in cats immunized with an avirulent feline infectious peritonitis virus (FIPV)-UCD1 and challenge-exposed with virulent FIPV-UCD8.

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Nucleotide sequence and expression of the feline vascular endothelial growth factor

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Vascular endothelial growth factor in the sera and effusions of patients with malignant and nonmalignant disease

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Serum levels of vascular endothelial growth factor (VEGF) are markedly elevated in patients with Wegener's granulomatosis.

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Increased serum levels of vascular endothelial growth factor in Kawasaki disease

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Role of vascular endothelial growth factor in ovarian cancer: inhibition of ascites formation by immunoneutralization

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Molecular cloning and sequence determination of the peplomer protein gene of feline infectious peritonitis virus type I.

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Comparison of the amino acid sequence and phylogenetic analysis of the peplomer, integral membrane and nucleocapsid proteins of feline, canine and porcine coronaviruses

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A review of feline infectious peritonitis virus infection: 1963-2008.

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Activation of p38 MAPK by feline infectious peritonitis virus regulates pro-inflammatory cytokine production in primary blood-derived feline mononuclear cells

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Isolation and culture of pulmonary endothelial cells

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Tumor necrosis factor-alpha promotes malignant pleural effusion

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B-cell activation in cats with feline infectious peritonitis (FIP) by FIP-virus-induced B-cell differentiation/survival factors

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7 January 2021

Neutrophil survival factors (TNF-alpha, GM-CSF, and G-CSF) produced by macrophages in cats infected with feline infectious peritonitis virus contribute to the pathogenesis of granulomatous lesions

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A "possible" involvement of TNF-alpha in apoptosis induction in peripheral blood lymphocytes of cats with feline infectious peritonitis

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TNF-alpha, produced by feline infectious peritonitis virus (FIPV)-infected macrophages, upregulates expression of type II FIPV receptor feline aminopeptidase N in feline macrophages

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Antibody-dependent enhancement occurs upon re-infection with the identical serotype virus in feline infectious peritonitis virus infection

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Elevated levels of plasma VEGF in patients with dengue hemorrhagic fever

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Feline Infectious Peritonitis Viruses Arise by Mutation from Endemic Feline Enteric Coronaviruses

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Monoclonal antibodies to the spike protein of feline infectious peritonitis virus mediate antibody-dependent enhancement of infection of feline macrophages

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Immunologic phenomena in the effusive form of feline infectious peritonitis

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Pathogenesis of feline infectious peritonitis: nature and development of viremia

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Activation of p38 MAPK and/or JNK contributes to increased levels of VEGF secretion in human malignant glioma cells

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Two related strains of feline infectious peritonitis virus isolated from immunocompromised cats infected with a feline enteric coronavirus

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Increased expression of vascular endothelial growth factor in pyogenic granulomas

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IL-6 activity in feline infectious peritonitis

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Identifiers

10.1016/J.VIRUSRES.2011.03.027

1 reference

Europe PubMed Central

PubMed publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:21473893 AND SRC:MED&resulttype=core&format=json

27 January 2020

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:21473893 AND SRC:MED&resulttype=core&format=json

27 January 2020

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