Disruption of growth hormone receptor gene causes diminished pancreatic islet size and increased insulin sensitivity in mice (Q44892368)

7 December 2017

title

Disruption of growth hormone receptor gene causes diminished pancreatic islet size and increased insulin sensitivity in mice (English)

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7 December 2017

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6

John J Kopchick

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2

Karen T Coschigano

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Jun-Li Liu

1

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Katie Robertson

3

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Mark Lipsett

4

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Yubin Guo

5

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Ujendra Kumar

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Ye Lauren Liu

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publication date

11 May 2004

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American Journal of Physiology - Endocrinology and Metabolism

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published in

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volume

287

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issue

3

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page(s)

E405-13

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Bone Mineral, Histomorphometry, and Body Composition in Adults with Growth Hormone Receptor Deficiency

7 December 2017

cites work

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Compensatory alterations of insulin signal transduction in liver of growth hormone receptor knockout mice.

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Autocrine human growth hormone inhibits placental transforming growth factor-beta gene transcription to prevent apoptosis and allow cell cycle progression of human mammary carcinoma cells

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Is the Laron mouse an accurate model of Laron syndrome?

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IGF-I and GH post-receptor signaling mechanisms for pancreatic beta-cell replication

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Disproportional skeletal growth and markedly decreased bone mineral content in growth hormone receptor -/- mice.

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Overexpression of parathyroid hormone-related protein in the pancreatic islets of transgenic mice causes islet hyperplasia, hyperinsulinemia, and hypoglycemia

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Attenuation of insulin secretion by insulin-like growth factor 1 is mediated through activation of phosphodiesterase 3B.

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A mammalian model for Laron syndrome produced by targeted disruption of the mouse growth hormone receptor/binding protein gene (the Laron mouse)

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Identifiers

DOI

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7 December 2017

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