The epithelial-mesenchymal transition induced by keratinocyte growth conditions is overcome by E6 and E7 from HPV16, but not HPV8 and HPV38: characterization of global transcription profiles. (Q44553066)

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scientific article published on 26 April 2009

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English	The epithelial-mesenchymal transition induced by keratinocyte growth conditions is overcome by E6 and E7 from HPV16, but not HPV8 and HPV38: characterization of global transcription profiles.	scientific article published on 26 April 2009

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26 January 2020

The epithelial-mesenchymal transition induced by keratinocyte growth conditions is overcome by E6 and E7 from HPV16, but not HPV8 and HPV38: characterization of global transcription profiles (English)

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26 January 2020

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Marco De Andrea

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26 January 2020

Giovanna Chiorino

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26 January 2020

Marisa Gariglio

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26 January 2020

Michele Mondini

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26 January 2020

Francesca Gugliesi

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26 January 2020

Valentina Dell'Oste

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Valentina Dell'oste

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26 January 2020

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26 January 2020

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Barbara Azzimonti

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26 January 2020

Cinzia Borgogna

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26 January 2020

Maria Scatolini

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26 January 2020

Chiara Ghimenti

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26 January 2020

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26 January 2020

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26 January 2020

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26 January 2020

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26 January 2020

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26 January 2020

Skin human papillomavirus type 38 alters p53 functions by accumulation of deltaNp73.

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7 January 2021

HPV-associated skin disease.

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7 January 2021

The E6 oncoproteins from human betapapillomaviruses differentially activate telomerase through an E6AP-dependent mechanism and prolong the lifespan of primary keratinocytes

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https://api.crossref.org/works/10.1016/J.VIROL.2009.03.033

7 January 2021

The E6 and E7 proteins of the cutaneous human papillomavirus type 38 display transforming properties

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https://api.crossref.org/works/10.1016/J.VIROL.2009.03.033

7 January 2021

Alpha- and betapapillomavirus E6/E7 genes differentially modulate pro-inflammatory gene expression

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https://api.crossref.org/works/10.1016/J.VIROL.2009.03.033

7 January 2021

Altered expression of UVB-induced cytokines in human papillomavirus-immortalized epithelial cells

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7 January 2021

Microarray analysis identifies differentiation-associated genes regulated by human papillomavirus type 16 E6.

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https://api.crossref.org/works/10.1016/J.VIROL.2009.03.033

7 January 2021

Human papillomaviruses: targeting differentiating epithelial cells for malignant transformation

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https://api.crossref.org/works/10.1016/J.VIROL.2009.03.033

7 January 2021

Betapapillomaviruses: innocent bystanders or causes of skin cancer

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https://api.crossref.org/works/10.1016/J.VIROL.2009.03.033

7 January 2021

Impairment of the telomere/telomerase system and genomic instability are associated with keratinocyte immortalization induced by the skin human papillomavirus type 38.

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https://api.crossref.org/works/10.1016/J.VIROL.2009.03.033

7 January 2021

Genomic and expression analysis of the 3q25-q26 amplification unit reveals TLOC1/SEC62 as a probable target gene in prostate cancer.

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7 January 2021

A method for protein assay in Laemmli buffer

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7 January 2021

Both Rb/p16INK4a inactivation and telomerase activity are required to immortalize human epithelial cells.

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https://api.crossref.org/works/10.1016/J.VIROL.2009.03.033

7 January 2021

The epithelial-mesenchymal transition: new insights in signaling, development, and disease

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7 January 2021

Genes involved in senescence and immortalization

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7 January 2021

Papillomavirus type 16 oncogenes downregulate expression of interferon-responsive genes and upregulate proliferation-associated and NF-kappaB-responsive genes in cervical keratinocytes

1 reference

https://api.crossref.org/works/10.1016/J.VIROL.2009.03.033

7 January 2021

Human papillomaviruses and non-melanoma skin cancer: basic virology and clinical manifestations

1 reference

https://api.crossref.org/works/10.1016/J.VIROL.2009.03.033

7 January 2021

Telomerase activation by human papillomavirus type 16 E6 protein: induction of human telomerase reverse transcriptase expression through Myc and GC-rich Sp1 binding sites

1 reference

https://api.crossref.org/works/10.1016/J.VIROL.2009.03.033

7 January 2021

In vitro characterization of an artificial dermal scaffold

1 reference

https://api.crossref.org/works/10.1016/J.VIROL.2009.03.033

7 January 2021

Growth and differentiation of human keratinocytes without a feeder layer or conditioned medium

1 reference

https://api.crossref.org/works/10.1016/J.VIROL.2009.03.033

7 January 2021

Transcriptional profiling of epidermal differentiation

1 reference

https://api.crossref.org/works/10.1016/J.VIROL.2009.03.033

7 January 2021

Putative telomere-independent mechanisms of replicative aging reflect inadequate growth conditions

1 reference

https://api.crossref.org/works/10.1016/J.VIROL.2009.03.033

7 January 2021

Growth kinetics and differentiation in vitro of normal human uroepithelial cells on collagen gel substrates in defined medium

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https://api.crossref.org/works/10.1016/J.VIROL.2009.03.033

7 January 2021

Serial cultivation of strains of human epidermal keratinocytes: the formation of keratinizing colonies from single cells

1 reference

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7 January 2021

A two-stage, p16(INK4A)- and p53-dependent keratinocyte senescence mechanism that limits replicative potential independent of telomere status

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7 January 2021

Biological functions of the small leucine-rich proteoglycans: from genetics to signal transduction.

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Development of skin tumors in mice transgenic for early genes of human papillomavirus type 8.

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7 January 2021

HPV-mediated cervical carcinogenesis: concepts and clinical implications

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7 January 2021

The human papillomavirus type 16 E6 and E7 oncoproteins dissociate cellular telomerase activity from the maintenance of telomere length

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7 January 2021

Specific betapapillomaviruses associated with squamous cell carcinoma of the skin inhibit UVB-induced apoptosis of primary human keratinocytes

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7 January 2021

Complex networks orchestrate epithelial-mesenchymal transitions

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7 January 2021

Transcriptional activation of the telomerase hTERT gene by human papillomavirus type 16 E6 oncoprotein

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7 January 2021

Immortalization of normal human embryonic fibroblasts by introduction of either the human papillomavirus type 16 E6 or E7 gene alone

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