Biological clock dysfunction exacerbates contact hypersensitivity in mice (Q44544254)

4 December 2017

title

Biological clock dysfunction exacerbates contact hypersensitivity in mice (English)

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2,4,6-Trinitrochlorobenzene

4 December 2017

main subject

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ChEBI release 2020-05-28

E Takita

1

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4 December 2017

S Yokota

2

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4 December 2017

Y Tahara

3

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A Hirao

4

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4 December 2017

N Aoki

5

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4 December 2017

Y Nakamura

6

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A Nakao

7

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S Shibata

8

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4 December 2017

language of work or name

English

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publication date

2 November 2012

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British Journal of Dermatology

4 December 2017

published in

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4 December 2017

volume

168

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4 December 2017

issue

1

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4 December 2017

page(s)

39-46

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Mammalian clock gene Cryptochrome regulates arthritis via proinflammatory cytokine TNF-alpha.

4 December 2017

cites work

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Immunological mechanisms of contact hypersensitivity in mice

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Toll-like receptor and IL-12 signaling control susceptibility to contact hypersensitivity

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Th2 cytokines, IgE and mast cells play a crucial role in the induction of para-phenylenediamine-induced contact hypersensitivity in mice

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Signal transducer and activator of transcription 6 is essential in the induction of contact hypersensitivity

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The crosstalk between physiology and circadian clock proteins

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Functional central rhythmicity and light entrainment, but not liver and muscle rhythmicity, are Clock independent

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Clock-dependent and independent transcriptional control of the two isoforms from the mouse Rorgamma gene.

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New insights into atopic dermatitis

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Effects of suprachiasmatic transplants on circadian rhythms of neuroendocrine function in golden hamsters

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Loss of a circadian adrenal corticosterone rhythm following suprachiasmatic lesions in the rat.

21 January 2018

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Clock mutation affects circadian regulation of circulating blood cells.

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Diurnal amplitudes of arterial pressure and heart rate are dampened in Clock mutant mice and adrenalectomized mice

21 January 2018

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Macrophages and neutrophils are the targets for immune suppression by glucocorticoids in contact allergy

21 January 2018

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Mast cells control neutrophil recruitment during T cell-mediated delayed-type hypersensitivity reactions through tumor necrosis factor and macrophage inflammatory protein 2.

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Local glucocorticoid production in the mouse lung is induced by immune cell stimulation

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Molecular mechanisms of glucocorticoids in the control of inflammation and lymphocyte apoptosis

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Circadian clock genes: non-circadian roles in sleep, addiction, and psychiatric disorders?

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Inhibition of pulmonary eosinophilia and airway hyperresponsiveness in allergic mice by rolipram: involvement of endogenously released corticosterone and catecholamines.

21 January 2018

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21 January 2018

Identifiers

DOI

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4 December 2017

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