The anti-inflammatory sesquiterpene lactone parthenolide suppresses CD95-mediated activation-induced-cell-death in T-cells (Q44196945)

30 November 2017

title

The anti-inflammatory sesquiterpene lactone parthenolide suppresses CD95-mediated activation-induced-cell-death in T-cells (English)

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30 November 2017

main subject

sesquiterpene lactone

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Li-Weber M

1

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30 November 2017

Giaisi M

2

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30 November 2017

Baumann S

3

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30 November 2017

Treiber MK

4

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30 November 2017

Krammer PH

5

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30 November 2017

publication date

1 November 2002

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Cell Death & Differentiation

30 November 2017

published in

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30 November 2017

volume

9

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30 November 2017

issue

11

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30 November 2017

page(s)

1256-1265

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Apoptosis by death factor

30 November 2017

cites work

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Stress-induced Fas ligand expression in T cells is mediated through a MEK kinase 1-regulated response element in the Fas ligand promoter

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DNA damaging agents induce expression of Fas ligand and subsequent apoptosis in T lymphocytes via the activation of NF-kappa B and AP-1.

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Activation interferes with the APO-1 pathway in mature human T cells.

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The death of a T-cell: expression of the CD95 ligand

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Autocrine T-cell suicide mediated by APO-1/(Fas/CD95)

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Ethnobotany and its role in drug development

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Sesquiterpene lactones specifically inhibit activation of NF-kappa B by preventing the degradation of I kappa B-alpha and I kappa B-beta

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Cysteine 38 in p65/NF-kappaB plays a crucial role in DNA binding inhibition by sesquiterpene lactones

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Activators and target genes of Rel/NF-kappaB transcription factors

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7 January 2021

T cell activation-induced and HIV tat-enhanced CD95(APO-1/Fas) ligand transcription involves NF-kappaB

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7 January 2021

Induction of apoptosis in uninfected lymphocytes by HIV-1 Tat protein

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7 January 2021

Sensitization of T cells to CD95-mediated apoptosis by HIV-1 Tat and gp120.

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7 January 2021

Activation-dependent transcriptional regulation of the human Fas promoter requires NF-kappaB p50-p65 recruitment

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Inhibition of the NF-kappa B transcription factor increases Bax expression in cancer cell lines

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Novel Egr/NF-AT composite sites mediate activation of the CD95 (APO-1/Fas) ligand promoter in response to T cell stimulation

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7 January 2021

Two NFAT Transcription Factor Binding Sites Participate in the Regulation of CD95 (Fas) Ligand Expression in Activated Human T Cells

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Transcriptional Regulation of the Human FasL Promoter-Enhancer Region

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7 January 2021

Cyclosporin A-sensitive transcription factor Egr-3 regulates Fas ligand expression

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7 January 2021

Role of Egr-2 in up-regulation of Fas ligand in normal T cells and aberrant double-negative lpr and gld T cells

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7 January 2021

Regulation of IkappaBbeta degradation. Similarities to and differences from IkappaBalpha

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7 January 2021

Sesquiterpene lactone containing Mexican Indian medicinal plants and pure sesquiterpene lactones as potent inhibitors of transcription factor NF-kappaB.

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7 January 2021

NF-kappa B RelA (p65) is essential for TNF-alpha-induced fas expression but dispensable for both TCR-induced expression and activation-induced cell death

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7 January 2021

Intracellular oxidation/reduction status in the regulation of transcription factors NF-kappaB and AP-1.

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7 January 2021

NF-kappaB as a primary regulator of the stress response

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7 January 2021

Control of apoptosis by Rel/NF-kappaB transcription factors

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7 January 2021

Monoclonal antibody-mediated tumor regression by induction of apoptosis.

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7 January 2021

Real time quantitative PCR

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7 January 2021

Structure of the human APO-1 gene

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7 January 2021

Regulation of p53 activity by its interaction with homeodomain-interacting protein kinase-2

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7 January 2021

Identifiers

DOI

10.1038/SJ.CDD.4401102

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30 November 2017

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