Receptor for advanced glycation endproducts (RAGE) is a key regulator of oval cell activation and inflammation-associated liver carcinogenesis in mice. (Q43506762)

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scientific article published in July 2013

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English	Receptor for advanced glycation endproducts (RAGE) is a key regulator of oval cell activation and inflammation-associated liver carcinogenesis in mice.	scientific article published in July 2013

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26 February 2020

Receptor for advanced glycation endproducts (RAGE) is a key regulator of oval cell activation and inflammation-associated liver carcinogenesis in mice (English)

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26 February 2020

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26 February 2020

Angelika Bierhaus

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Angelika Bierhaus

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26 February 2020

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Tobias Pusterla

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26 February 2020

Julia Nèmeth

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26 February 2020

Ilan Stein

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26 February 2020

Lars Wiechert

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26 February 2020

Silke Marhenke

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26 February 2020

Thomas Longerich

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26 February 2020

Varun Kumar

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26 February 2020

Bernd Arnold

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26 February 2020

Arndt Vogel

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26 February 2020

Eli Pikarsky

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26 February 2020

Jochen Hess

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26 February 2020

Peter Angel

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26 February 2020

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1 July 2013

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26 February 2020

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26 February 2020

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26 February 2020

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26 February 2020

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26 February 2020

S100A8/9 induces cell death via a novel, RAGE-independent pathway that involves selective release of Smac/DIABLO and Omi/HtrA2.

1 reference

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S100A8 and S100A9 mediate endotoxin-induced cardiomyocyte dysfunction via the receptor for advanced glycation end products

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HMGB1 and RAGE in inflammation and cancer

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Multiple levels of regulation determine the role of the receptor for AGE (RAGE) as common soil in inflammation, immune responses and diabetes mellitus and its complications

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The receptor RAGE: Bridging inflammation and cancer

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RAGE signaling sustains inflammation and promotes tumor development

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RAGE, carboxylated glycans and S100A8/A9 play essential roles in colitis-associated carcinogenesis

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21 January 2018

Blockade of receptor for advanced glycation end product (RAGE) attenuates ischemia and reperfusion injury to the liver in mice

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21 January 2018

Receptor for advanced glycation end product (RAGE)-dependent modulation of early growth response-1 in hepatic ischemia/reperfusion injury

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RAGE limits regeneration after massive liver injury by coordinated suppression of TNF-alpha and NF-kappaB

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21 January 2018

The nuclear factor HMGB1 mediates hepatic injury after murine liver ischemia-reperfusion

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High Mobility Group Box 1 Release from Hepatocytes during Ischemia and Reperfusion Injury Is Mediated by Decreased Histone Deacetylase Activity

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HMGB1 cytoplasmic translocation in patients with acute liver failure

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Specific siRNA targeting the receptor for advanced glycation end products inhibits experimental hepatic fibrosis in rats

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A novel function of the receptor for advanced glycation end-products (RAGE) in association with tumorigenesis and tumor differentiation of HCC

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High-mobility Group Box 1 is Associated with Clinicopathologic Features in Patients with Hepatocellular Carcinoma

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The expression of HMGB1 protein and its receptor RAGE in human malignant tumors

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21 January 2018

What is the role of the receptor for advanced glycation end products-ligand axis in liver injury?

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Liver stem cells: implications for hepatocarcinogenesis

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NF-kappaB functions as a tumour promoter in inflammation-associated cancer

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Receptor for advanced glycation end products (RAGE) regulates sepsis but not the adaptive immune response

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A modified choline-deficient, ethionine-supplemented diet protocol effectively induces oval cells in mouse liver

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Immunity, Inflammation, and Cancer

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Contradictory functions of NF-kappaB in liver physiology and cancer

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Cell proliferation and hepatocarcinogenesis in rat initiated by diethylnitrosamine and promoted by phenobarbital: potential roles of early DNA damage and liver metallothionein expression.

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A lymphotoxin-driven pathway to hepatocellular carcinoma

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Dose- and time-dependent oval cell reaction in acetaminophen-induced murine liver injury.

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Liver inflammation and cytokine production, but not acute phase protein synthesis, accompany the adult liver progenitor (oval) cell response to chronic liver injury

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Isolation, culture and immortalisation of hepatic oval cells from adult mice fed a choline-deficient, ethionine-supplemented diet

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Bioenergetic differences selectively sensitize tumorigenic liver progenitor cells to a new gold(I) compound

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Molecular viral oncology of hepatocellular carcinoma

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Multiple adaptive mechanisms to chronic liver disease revealed at early stages of liver carcinogenesis in the Mdr2-knockout mice

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Toll-like Receptor 4 and Hepatic Fibrogenesis

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Advanced glycation end products (AGEs) and their involvement in liver disease

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Liver cancer stem cells

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A rodent model of NASH with cirrhosis, oval cell proliferation and hepatocellular carcinoma

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Impaired preneoplastic changes and liver tumor formation in tumor necrosis factor receptor type 1 knockout mice

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Disruption of Trp53 in livers of mice induces formation of carcinomas with bilineal differentiation.

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A novel prognostic subtype of human hepatocellular carcinoma derived from hepatic progenitor cells

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Kupffer cell heterogeneity: functional properties of bone marrow derived and sessile hepatic macrophages

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The lack of chromosomal protein Hmg1 does not disrupt cell growth but causes lethal hypoglycaemia in newborn mice

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Blockade of RAGE–amphoterin signalling suppresses tumour growth and metastases

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Blockade of the receptor for advanced glycation end products attenuates acetaminophen-induced hepatotoxicity in mice

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Is HMGB1 a New Indirect Marker for Revealing Fibrosis in Chronic Hepatitis and a New Therapeutic Target in Treatment?

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Common antigens of mouse oval and biliary epithelial cells. Expression on newly formed hepatocytes

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RAGE and ICAM-1 cooperate in mediating leukocyte recruitment during acute inflammation in vivo

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