Insulin attenuates myocardial ischemia/reperfusion injury via reducing oxidative/nitrative stress. (Q43177442)

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17 January 2020

title

Insulin attenuates myocardial ischemia/reperfusion injury via reducing oxidative/nitrative stress (English)

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17 January 2020

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17 January 2020

author name string

Lele Ji

1

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17 January 2020

Lihua Zhang

3

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17 January 2020

Wenchong Liu

4

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17 January 2020

Xiaoqing Cai

5

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17 January 2020

Lei Zhang

6

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17 January 2020

Qiangsun Zheng

7

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17 January 2020

Haifeng Zhang

8

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17 January 2020

Feng Gao

9

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17 January 2020

publication date

2 February 2010

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American Journal of Physiology - Endocrinology and Metabolism

17 January 2020

published in

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17 January 2020

volume

298

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17 January 2020

issue

4

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17 January 2020

page(s)

E871-80

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Apparent hydroxyl radical production by peroxynitrite: implications for endothelial injury from nitric oxide and superoxide

17 January 2020

cites work

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Peroxynitrite-induced alpha-actinin nitration and contractile alterations in isolated human myocardial cells

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Status of myocardial antioxidants in ischemia-reperfusion injury

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Suppression of apoptosis by nitric oxide via inhibition of interleukin-1beta-converting enzyme (ICE)-like and cysteine protease protein (CPP)-32-like proteases

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Nitric oxide, superoxide, and peroxynitrite in myocardial ischaemia-reperfusion injury and preconditioning

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Nitric oxide mediates the antiapoptotic effect of insulin in myocardial ischemia-reperfusion: the roles of PI3-kinase, Akt, and endothelial nitric oxide synthase phosphorylation

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Role of TNF-alpha-induced reactive oxygen species in endothelial dysfunction during reperfusion injury

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Opioid-induced cardioprotection occurs via glycogen synthase kinase beta inhibition during reperfusion in intact rat hearts

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INO-4885 [5,10,15,20-tetra[N-(benzyl-4'-carboxylate)-2-pyridinium]-21H,23H-porphine iron(III) chloride], a peroxynitrite decomposition catalyst, protects the heart against reperfusion injury in mice

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Reciprocal relationships between insulin resistance and endothelial dysfunction: molecular and pathophysiological mechanisms.

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Nitric oxide as a bifunctional regulator of apoptosis

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Intensive insulin therapy protects the endothelium of critically ill patients

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Peroxynitrite is a major trigger of cardiomyocyte apoptosis in vitro and in vivo

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Physiological mechanisms regulating the expression of endothelial-type NO synthase.

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Adiponectin improves endothelial function in hyperlipidemic rats by reducing oxidative/nitrative stress and differential regulation of eNOS/iNOS activity

21 January 2018

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Critical timing of L-arginine treatment in post-ischemic myocardial apoptosis-role of NOS isoforms

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Vasculoprotective effect of insulin in the ischemic/reperfused canine heart: role of Akt-stimulated NO production

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Prospective randomised study of intensive insulin treatment on long term survival after acute myocardial infarction in patients with diabetes mellitus. DIGAMI (Diabetes Mellitus, Insulin Glucose Infusion in Acute Myocardial Infarction) Study Group

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Myocardial ischemia/reperfusion-injury, a clinical view on a complex pathophysiological process

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Lactate and PO2 modulate superoxide anion production in bovine cardiac myocytes: potential role of NADH oxidase

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Nitrosative stress and pharmacological modulation of heart failure

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21 January 2018

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Peroxynitrite-induced myocardial injury is mediated through matrix metalloproteinase-2.

21 January 2018

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Insulin inhibits myocardial ischemia-induced apoptosis and alleviates chronic adverse changes in post-ischemic cardiac structure and function.

21 January 2018

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The role of inflammatory cytokines in endothelial dysfunction

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Role of insulin in the anti-apoptotic effect of glucose-insulin-potassium in rabbits with acute myocardial ischemia and reperfusion

21 January 2018

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21 January 2018

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